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Volumn 398, Issue 1, 2002, Pages 132-140

A calmodulin binding site in the tuberous sclerosis 2 gene product is essential for regulation of transcription events and is altered by mutations linked to tuberous sclerosis and lymphangioleiomyomatosis

(4) Noonan, Daniel J a Lou, Dingyuan a Griffith, Nicole a Vanaman, Thomas C a

a UNIVERSITY OF KENTUCKY (United States)

Author keywords

[No Author keywords available]

Indexed keywords

CALMODULIN; CALMODULIN BINDING PROTEIN; CELL NUCLEUS RECEPTOR; GENE PRODUCT; PHOSPHODIESTERASE; SEPHAROSE; STEROID RECEPTOR; SYNTHETIC PEPTIDE; TRYPTOPHAN; TUBERIN;

ARTICLE; BACTERIOPHAGE LAMBDA; BINDING SITE; CARBOXY TERMINAL SEQUENCE; DNA LIBRARY; EMBRYO; ENZYME ACTIVATION; FLUORESCENCE; GENE; GENE ACTIVITY; GENE DELETION; GENE MUTATION; GENETIC LINKAGE; HUMAN; HUMAN CELL; LYMPHANGIOLEIOMYOMATOSIS; PRIORITY JOURNAL; PROTEIN DOMAIN; SIGNAL TRANSDUCTION; TRANSCRIPTION REGULATION; TSC2 GENE; TUBEROUS SCLEROSIS; WESTERN BLOTTING;

EID: 0036478805 PISSN: 00039861 EISSN: None Source Type: Journal
DOI: 10.1006/abbi.2001.2682 Document Type: Article

Times cited : (48)

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