Transient and sustained ERK phosphorylation and nuclear translocation in growth control

Journal of Cellular Physiology

Volumn 192, Issue 2, 2002, Pages 151-159

  Adachi, Takahito ^a   Kar, Siddhartha ^a   Wang, Meifang ^a   Carr, Brian I ^a  

^a UNIVERSITY OF PITTSBURGH MEDICAL CENTER   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

1,4 DIAMINO 1,4 BIS(2 AMINOPHENYLTHIO) 2,3 DICYANOBUTADIENE; 1,4 NAPHTHOQUINONE DERIVATIVE; 2 (2 HYDROXYETHYLSULFANYL) 3 METHYL 1,4 NAPHTHOQUINONE; EPIDERMAL GROWTH FACTOR; EPIDERMAL GROWTH FACTOR RECEPTOR; MITOGEN ACTIVATED PROTEIN KINASE INHIBITOR; PD 098056; PROTEIN TYROSINE PHOSPHATASE INHIBITOR; UNCLASSIFIED DRUG;

ARTICLE; CONTROLLED STUDY; DRUG MECHANISM; DRUG RECEPTOR BINDING; ENZYME ACTIVITY; GROWTH INHIBITION; GROWTH REGULATION; GROWTH STIMULATION; HEPATOMA CELL; HUMAN; HUMAN CELL; PRIORITY JOURNAL; PROTEIN PHOSPHORYLATION;

ACTIVE TRANSPORT, CELL NUCLEUS; CELL DIVISION; CELL NUCLEUS; DNA-BINDING PROTEINS; DRUG INTERACTIONS; ENZYME INHIBITORS; EPIDERMAL GROWTH FACTOR; ETS-DOMAIN PROTEIN ELK-1; GROWTH INHIBITORS; HUMANS; MITOGEN-ACTIVATED PROTEIN KINASE KINASES; MITOGEN-ACTIVATED PROTEIN KINASES; PHOSPHORIC MONOESTER HYDROLASES; PHOSPHORYLATION; PROTEIN TRANSPORT; PROTO-ONCOGENE PROTEINS; RECEPTOR, EPIDERMAL GROWTH FACTOR; TRANSCRIPTION FACTORS; TUMOR CELLS, CULTURED; VITAMIN K;

CERVUS ELAPHUS;

EID: 0036306889     PISSN: 00219541     EISSN: None     Source Type: Journal    
DOI: 10.1002/jcp.10124     Document Type: Article

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