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Volumn 267, Issue 15, 2000, Pages 4676-4684
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Expression of immediate early gene pip92 during anisomycin-induced cell death is mediated by the JNK- and p38-dependent activation of Elk1
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Author keywords
Anisomycin; Cell death; Elkl; MAP kinase; Pip92
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Indexed keywords
ANISOMYCIN;
IMMEDIATE EARLY PROTEIN;
IMMEDIATE EARLY PROTEIN PIP92;
MITOGEN ACTIVATED PROTEIN KINASE;
TRANSCRIPTION FACTOR;
TRANSCRIPTION FACTOR ELK;
UNCLASSIFIED DRUG;
ANIMAL CELL;
ARTICLE;
CELL DEATH;
CELL STRAIN 3T3;
GENE ACTIVATION;
MOUSE;
NONHUMAN;
PRIORITY JOURNAL;
PROTEIN EXPRESSION;
PROTEIN PHOSPHORYLATION;
SERUM RESPONSIVE ELEMENT;
TRANSACTIVATION;
3T3 CELLS;
ANIMALS;
ANISOMYCIN;
BLOTTING, NORTHERN;
CELL DEATH;
CHLORAMPHENICOL O-ACETYLTRANSFERASE;
DNA-BINDING PROTEINS;
DOSE-RESPONSE RELATIONSHIP, DRUG;
ENZYME ACTIVATION;
ETS-DOMAIN PROTEIN ELK-1;
FUNGAL PROTEINS;
GENE DELETION;
IMMEDIATE-EARLY PROTEINS;
MICE;
MITOGEN-ACTIVATED PROTEIN KINASE 8;
MITOGEN-ACTIVATED PROTEIN KINASES;
P38 MITOGEN-ACTIVATED PROTEIN KINASES;
PHOSPHORYLATION;
POTASSIUM CHANNELS;
PRECIPITIN TESTS;
PROMOTER REGIONS (GENETICS);
PROTEIN BIOSYNTHESIS;
PROTEIN SYNTHESIS INHIBITORS;
PROTEINS;
PROTO-ONCOGENE PROTEINS;
RECOMBINANT FUSION PROTEINS;
RNA;
SACCHAROMYCES CEREVISIAE PROTEINS;
SIGNAL TRANSDUCTION;
THYMIDINE KINASE;
TIME FACTORS;
TRANS-ACTIVATION (GENETICS);
TRANSCRIPTION FACTORS;
TRANSFECTION;
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EID: 0033861869
PISSN: 00142956
EISSN: None
Source Type: Journal
DOI: 10.1046/j.1432-1327.2000.01517.x Document Type: Article |
Times cited : (36)
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References (46)
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