Endothelial damage during myocardial preservation and storage

Annals of Thoracic Surgery

Volumn 73, Issue 2, 2002, Pages 682-690

  Parolari, Alessandro ^a   Rubini, Patrizia ^b   Cannata, Aldo ^b   Bonati, Luigi ^b   Alamanni, Francesco ^b   Tremoli, Elena ^b   Biglioli, Paolo ^b  

^a UNIVERSITY OF MILAN   (Italy)

^b NONE

Author keywords

[No Author keywords available]

Indexed keywords

CELL PROTECTION; CORONARY ARTERY BLOOD FLOW; HEART ARREST; HEART FUNCTION; HEART MUSCLE CELL; HEART MUSCLE ISCHEMIA; HEART MUSCLE REPERFUSION; HEART SURGERY; HEART TRANSPLANTATION; ISCHEMIA; METHODOLOGY; ORGAN PRESERVATION; PRIORITY JOURNAL; REVIEW; TISSUE INJURY; VASCULAR ENDOTHELIUM;

EID: 0036159668     PISSN: 00034975     EISSN: None     Source Type: Journal    
DOI: 10.1016/S0003-4975(01)03029-6     Document Type: Article

References (89)

1. Relative susceptibility of endothelium and myocardial cells to ischaemia-reperfusion injury
2. Role of endothelial cells in transplantation
3. Cardiac allograft vasculopathy: The central pathogenetic role of ischemia-induced endothelial cell injury
4. Chronic rejection in human heart transplantation
5. Human coronary transplantation-associated arteriosclerosis. Evidence for a chronic immune reaction to activated graft endothelial cells
6. Mechanisms of hypoxia-induced endothelial cell death. Role of p53 in apoptosis
7. Hypoxia modulates the barrier and coagulant function of cultured bovine endothelium
8. Tumor necrosis factor-α enhances hypoxia-reoxygenation-mediated apoptosis in cultured human coronary artery endothelial cells: Critical role of protein kinase
9. Endothelial cell tolerance to hypoxia. Potential role of purine nucleotide phosphatases
10. Hypoxia-mediated induction of endothelial cell interleukin-1 alpha. An autocrine mechanism promoting expression of leukocyte adhesion molecules on the vessel surface
11. Hypoxic induction of interleukin-8 gene expression in human endothelial cells
12. Free radicals and myocardial protection: A surgical viewpoint
13. Prolonged hypoxia alters endothelial barrier function
14. Time course and mechanisms of endothelial dysfunction in isolated ischemic and hypoxic perfused rat hearts
15. Hydrogen peroxide stimulates the synthesis of platelet-activating factor by endothelium and induces endothelial cell-dependent neutrophil adhesion
16. Tissue destruction by neutrophils
17. Treating myocardial ischemia-reperfusion injury, by targeting endothelial cell transcription
18. α
19. An essential role for NF-κB in the cardioadaptive response to ischemia
20. Inhibition of nuclear factor-kappa B nuclear localization reduces human E-selectin expression and the systemic inflammatory response
21. Hypothermia abolishes hypoxia-induced hyperpermeability in brain microvessel endothelial cells
22. Hypothermia reversibly inhibits endothelial cell expression of E-selectin and tissue factor
23. Hypothermia inhibits human E-selectin transcription
24. The effects of hypothermia on myocardial oxygen consumption and transmural coronary blood flow in the potassium arrested heart
25. - in cultured human endothelial cells
26. Changes in intracellular calcium induced by acute hypothermia in parenchymal, endothelial, and Kupffer cells of the rat liver
27. Postcardioplegia acute cardiac dysfunction and reperfusion injury
28. Hypothermia during cardiopulmonary bypass delays but does not prevent neutrophil-endothelial cell adhesion. A clinical study
29. Effects of depolarizing or non-depolarizing preservation solutions on human endothelial cells during cold hypoxia
30. Cold-induced apoptosis in cultured hepatocytes and liver endothelial cells: Mediation by reactive oxygen species
31. Biomechanical activation: An emerging paradigm in endothelial adhesion biology
32. The role of rheology in atherosclerotic coronary artery disease
33. Blood flow-induced remodeling of the artery wall
34. Effects of pressure and duration of hyperkalemic infusions on endothelial function
35. Effect of cardioplegia infusion pressure on coronary artery endothelium and cardiac mechanical function
36. Crystalloid cardioplegia and hypothermia do not impair endothelium-dependent relaxation or damage vascular smooth muscle of epicardial coronary arteries
37. High cardioplegic perfusion pressure entails reduced myocardial recovery
38. Delivery pressure of the cardioplegic solution influences myocardial protection
39. Rheologic considerations in organ preservation
40. Long term preservation of the heart: The effect of infusion pressure during continuous hypothermic cardioplegia
41. Effects of pulsatile perfusion pressure and storage on hearts preserved for 24 hours under hypothermia, for transplantation
42. Organ preservation solutions in heart transplantation-patterns of usage and related survival
43. Which organ preservation solution best protects vascular endothelium?
44. Trehalose-containing solutions enhance preservation of cultured endothelial cells
45. Protective effects of various preservation solutions on cultured endothelial cells
46. Prolonged hypothermic cardiac storage with University of Wisconsin solution. An assessment with human cell cultures
47. Endothelial cell toxicity of solid-organ preservation solutions
48. Endothelial cell preservation using organ storage solutions
49. University of Wisconsin solution provides superior myocardial preservation compared with Stanford cardioplegic solution
50. Paradoxical cholinergic and purinergic vascular reactivity of rabbit thoracic aorta cold-stored in University of Wisconsin solution
51. Functional and morphological assessment of rat aorta stored in University of Wisconsin and Euro-Collins solutions
52. Ultrastructural analysis of human endothelial cells after hypothermic storage in organ preservation solutions
53. Apoptosis is involved in endothelial cell damage during preservation and influenced by organ storage solutions
54. Organ preservation solutions increase endothelial permeability and promote loss of junctional proteins
55. Prolonged hypothermic cardiac storage with histidinetryptophan-ketoglutarate solution: Comparison with glucose-insulin-potassium and University of Wisconsin solutions
56. HTK solution is more effective than UW solution for cardiac preservation
57. HTK versus UW solution for myocardial protection during moderate hypothermia
58. The role of preservation solutions in coronary endothelial damage during cold storage
59. Heart preservation in HTK solution: Role of coronary vasculature in recovery of cardiac function
60. Cardioprotective effect of nicorandil in histidine-tryptophan-ketoglutarate solution during the cold storage of isolated hearts
61. Superior qualities of University of Wisconsin solution for ex vivo preservation of the pig heart
62. Long-term preservation of vascular endothelium and smooth muscle
63. Preservation of endothelium-dependent vasodilation with low-potassium University of Wisconsin solution
64. Respiratory defect as an early event in preservation-reoxygenation injury of endothelial cells
65. Comparison of University of Wisconsin and St. Thomas' Hospital solutions on endothelium-derived hyperpolarizing factor-mediated function in coronary microarteries
66. Hypothermic storage with University of Wisconsin solution preserves endothelial and vascular smooth muscle function
67. Functional assessment of rat aorta after cold storage in different media
68. Altered endothelial and smooth muscle cell reactivity caused by University of Wisconsin preservation solution in human saphenous vein
69. Impairment of vascular endothelial function by high-potassium storage solutions
70. Coronary vascular responsiveness to 5-hydroxytryptamine before and after infusion of hyperkalemic crystalloid cardioplegic solution in the rat heart. Possible evidence of endothelial damage
71. Functional endothelial damage by high potassium cardioplegic solutions to saphenous vein bypass grafts
72. Hyperkalemia exposure impairs EDHF-mediated endothelial function in the human coronary artery
73. Impaired endothelium-derived hyperpolarizing factor-mediated relaxation in coronary arteries by cold storage with University of Wisconsin solution
74. Hyperkalemia alters endothelium-dependent relaxation through non-nitric oxide and noncyclooxygenase pathway: A mechanism for coronary dysfunction due to cardioplegia
75. Hyperkalemia alters EDHF-mediated hyperpolarization, and relaxation in coronary arteries
76. Depolarizing cardiac arrest and endothelium-derived hyperpolarizing factor-mediated hyperpolarization and relaxation in coronary arteries: The effect and mechanism
77. Importance of endothelium-derived hyperpolarizing factor in human arteries
78. + channels
79. Protective effect of reduced glutathione on endothelial function of coronary arteries subjected to prolonged cold storage
80. Coronary endothelial dysfunction of isolated hearts subjected to prolonged cold storage: Patterns and contributing factors
81. Cytotoxicity of cardioplegic solutions: Evaluation by tissue culture
82. Cardioplegia and vascular injury. Dissociation of the effects of ischemia from those of the cardioplegic solution
83. Ischaemic endothelial dysfunction after single or multidose cardioplegia
84. Impaired endothelium-dependent relaxation after cardiac global ischemia and reperfusion: Role of warm blood cardioplegia
85. Blood and albumin cardioplegia preserve endothelium-dependent microvascular responses
86. Methods of cardiac preservation alter the function of the endothelium in porcine coronary arteries
87. Strategies and logic of cardioplegic delivery to prevent, avoid, and reverse ischemic and reperfusion damage
88. Mechanisms causing coronary microvascular dysfunction following crystalloid cardioplegia and reperfusion
89. Microcirculatory dysfunction following perfusion with hyperkalemic, hypothermic, cardioplegic solutions and blood reperfusion. Effect of adenosine