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Volumn 61, Issue 1, 2002, Pages 230-238
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Loss of nicotinic receptors in monkey striatum after 1-methyl-4-phenyl-1,2,3,6-tetrahydropyridine treatment is due to a decline in α-conotoxin MII sites
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Author keywords
[No Author keywords available]
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Indexed keywords
1,2,3,6 TETRAHYDRO 1 METHYL 4 PHENYLPYRIDINE;
3 (2 AZETIDINYLMETHOXY)PYRIDINE;
CHOLINERGIC RECEPTOR;
CONOTOXIN;
CYTISINE;
DOPAMINE TRANSPORTER;
EPIBATIDINE;
IODINE 125;
NICOTINE;
NICOTINIC RECEPTOR;
ANIMAL EXPERIMENT;
ANIMAL TISSUE;
ARTICLE;
BASAL GANGLION;
BINDING AFFINITY;
CAUDATE NUCLEUS;
CONTROLLED STUDY;
CORPUS STRIATUM;
DRUG BINDING;
FRONTAL CORTEX;
NONHUMAN;
PARKINSON DISEASE;
PARKINSONISM;
PRIORITY JOURNAL;
PROTEIN EXPRESSION;
STRIATONIGRAL DEGENERATION;
THALAMUS;
1-METHYL-4-PHENYL-1,2,3,6-TETRAHYDROPYRIDINE;
ANIMALS;
BICYCLO COMPOUNDS, HETEROCYCLIC;
BINDING, COMPETITIVE;
CAUDATE NUCLEUS;
CONOTOXINS;
CORPUS STRIATUM;
DOPAMINE AGENTS;
IODINE RADIOISOTOPES;
MOTOR ACTIVITY;
NICOTINIC AGONISTS;
PUTAMEN;
PYRIDINES;
RADIOLIGAND ASSAY;
RECEPTORS, NICOTINIC;
SAIMIRI;
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EID: 0036144163
PISSN: 0026895X
EISSN: None
Source Type: Journal
DOI: 10.1124/mol.61.1.230 Document Type: Article |
Times cited : (51)
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References (40)
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