A cycloheximide-sensitive factor regulates TCDD-induced degradation of the aryl hydrocarbon receptor

Chemosphere

Volumn 46, Issue 9-10, 2002, Pages 1491-1500

  Ma, Qiang ^a   Baldwin, Kimberly T ^a  

^a NATIONAL INSTITUTE FOR OCCUPATIONAL SAFETY AND HEALTH   (United States)

Author keywords

2,3,7,8 Tetrachlorodibenzo p dioxin; 26S proteasome; Ah receptor; Degradation; Superinduction

Indexed keywords

DEGRADATION; DNA; HALOGENATION; PROTEINS; SYNTHESIS (CHEMICAL);

TRANSCRIPTION ACTIVATION (TA);

AROMATIC HYDROCARBONS;

2,3,7,8 TETRACHLORODIBENZO PARA DIOXIN; AROMATIC HYDROCARBON RECEPTOR; CYCLOHEXIMIDE; PROTEASOME;

MEDICAL GEOGRAPHY;

ANIMAL CELL; ARTICLE; CONTROLLED STUDY; DNA BINDING; ELECTROPHORESIS; HALF LIFE TIME; IMMUNOPRECIPITATION; MOUSE; NONHUMAN; PROTEIN DEGRADATION; PROTEIN SYNTHESIS INHIBITION; REDUCTION; RNA ANALYSIS; TRANSCRIPTION INITIATION;

ANIMALS; CYCLOHEXIMIDE; CYTOCHROME P-450 CYP1A1; DNA; DOWN-REGULATION; ENVIRONMENTAL POLLUTANTS; GENE EXPRESSION REGULATION; HALF-LIFE; LIGANDS; MICE; PEPTIDE HYDROLASES; PROTEASOME ENDOPEPTIDASE COMPLEX; PROTEIN SYNTHESIS INHIBITORS; RECEPTORS, ARYL HYDROCARBON; TETRACHLORODIBENZODIOXIN;

EID: 0036124367     PISSN: 00456535     EISSN: None     Source Type: Journal    
DOI: 10.1016/S0045-6535(01)00270-3     Document Type: Article

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