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Volumn 119, Issue 6, 2002, Pages 511-520
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Volume-regulated anion channels serve as an auto/paracrine nucleotide release pathway in aortic endothelial cells
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Author keywords
ATP release; Calcium; Hypotonic stress; Nitric oxide; Volume regulated anion channel
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Indexed keywords
ADENOSINE;
ADENOSINE DIPHOSPHATE;
ADENOSINE TRIPHOSPHATE;
ANION CHANNEL;
CALCIUM ION;
CHLORIDE ION;
CYTIDINE TRIPHOSPHATE;
FLUOXETINE;
GLIBENCLAMIDE;
GUANOSINE TRIPHOSPHATE;
NUCLEOTIDE;
TAMOXIFEN;
URIDINE TRIPHOSPHATE;
VERAPAMIL;
ANION;
BREFELDIN A;
CHLORIDE;
HYPOTONIC SOLUTION;
ION CHANNEL;
NITRIC OXIDE;
PROTEIN SYNTHESIS INHIBITOR;
ANIMAL CELL;
AORTA;
ARTICLE;
AUTOCRINE EFFECT;
BINDING AFFINITY;
CALCIUM CELL LEVEL;
CELL MEMBRANE PERMEABILITY;
CELL TYPE;
CELL VOLUME;
CHANNEL GATING;
CONTROLLED STUDY;
DEPOLARIZATION;
ENDOTHELIUM CELL;
EXTRACELLULAR SPACE;
MECHANICAL STRESS;
NONHUMAN;
OSCILLATION;
PARACRINE SIGNALING;
PROTEIN BINDING;
PROTEIN SECRETION;
VASCULAR ENDOTHELIUM;
ANIMAL;
BIOLOGICAL MODEL;
CATTLE;
CELL MEMBRANE POTENTIAL;
CYTOLOGY;
DRUG EFFECT;
METABOLISM;
OSMOTIC PRESSURE;
PHYSIOLOGY;
THORACIC AORTA;
ADENOSINE DIPHOSPHATE;
ADENOSINE TRIPHOSPHATE;
ANIMAL;
ANIONS;
AORTA, THORACIC;
AUTOCRINE COMMUNICATION;
BREFELDIN A;
CATTLE;
CHLORIDES;
ENDOTHELIUM, VASCULAR;
HYPOTONIC SOLUTIONS;
ION CHANNELS;
MEMBRANE POTENTIALS;
MODELS, BIOLOGICAL;
NITRIC OXIDE;
OSMOTIC PRESSURE;
PARACRINE COMMUNICATION;
PROTEIN SYNTHESIS INHIBITORS;
SUPPORT, NON-U.S. GOV'T;
ANIMALS;
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EID: 0036016538
PISSN: 00221295
EISSN: None
Source Type: Journal
DOI: 10.1085/jgp.20028540 Document Type: Article |
Times cited : (130)
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References (42)
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