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Proceedings of the National Academy of Sciences of the United States of America

Volumn 98, Issue 5, 2001, Pages 2526-2531

Targeted disruption of the Kcnq1 gene produces a mouse model of Jervell and Lange-Nielsen Syndrome

(9) Casimiro, Mathew C a Knollmann, Björn C b Ebert, Steven N b Vary Jr , Jay C a,c Greene, Anne E d Franz, Michael R b Grinberg, Alexander a Huang, Sing Ping a Pfeifer, Karl a

a EUNICE KENNEDY SHRIVER NATIONAL INSTITUTE OF CHILD HEALTH AND HUMAN DEVELOPMENT (United States)

b GEORGETOWN UNIVERSITY MEDICAL CENTER (United States)

c NATIONAL INSTITUTES OF HEALTH (United States)

d CHILDREN'S NATIONAL MEDICAL CENTER (United States)

Author keywords

[No Author keywords available]

Indexed keywords

POTASSIUM CHANNEL;

ANIMAL MODEL; ANIMAL TISSUE; ARTICLE; GENE DISRUPTION; GENE MUTATION; GENE TARGETING; HEARING LOSS; HEART REPOLARIZATION; INNER EAR; LONG QT SYNDROME; MOUSE; NONHUMAN; P WAVE; PRIORITY JOURNAL; T WAVE; TRANSGENIC MOUSE;

ACTION POTENTIALS; ANIMALS; BASE SEQUENCE; DISEASE MODELS, ANIMAL; DNA PRIMERS; EAR, INNER; ELECTROCARDIOGRAPHY; HOMEOSTASIS; KCNQ POTASSIUM CHANNELS; KCNQ1 POTASSIUM CHANNEL; LONG QT SYNDROME; MICE; MICE, MUTANT STRAINS; MUTATION; PHENOTYPE; POTASSIUM CHANNELS; POTASSIUM CHANNELS, VOLTAGE-GATED;

ANIMALIA; MUS MUSCULUS; RODENTIA;

EID: 0035956935 PISSN: 00278424 EISSN: None Source Type: Journal
DOI: 10.1073/pnas.041398998 Document Type: Article

Times cited : (221)

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