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Volumn 430, Issue 1, 2001, Pages 59-68
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Characterization of the signal transduction pathways mediating morphine withdrawal-stimulated c-fos expression in hypothalamic nuclei
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Author keywords
C fos; Ca2+ channel; Morphine withdrawal; Paraventricular nucleus; Protein kinase A; Protein kinase C; Supraoptic nucleus
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Indexed keywords
CALCIUM;
CALCIUM CHANNEL BLOCKING AGENT;
CALCIUM CHANNEL L TYPE;
CALPHOSTIN C;
CHELERYTHRINE;
CYCLIC AMP DEPENDENT PROTEIN KINASE;
CYCLIC AMP DEPENDENT PROTEIN KINASE INHIBITOR;
MORPHINE;
N (2 GUANIDINOETHYL) 5 ISOQUINOLINESULFONAMIDE;
NALOXONE;
NIMODIPINE;
PROTEIN C FOS;
PROTEIN KINASE C;
PROTEIN KINASE C INHIBITOR;
ANIMAL EXPERIMENT;
ANIMAL MODEL;
ANIMAL TISSUE;
ANTERIOR HYPOTHALAMUS;
ARTICLE;
CALCIUM TRANSPORT;
CELL ACTIVATION;
CONTROLLED STUDY;
DRUG WITHDRAWAL;
ENZYME ACTIVE SITE;
ENZYME ACTIVITY;
IMMUNOREACTIVITY;
MALE;
NEUROSECRETORY CELL;
NONHUMAN;
PRIORITY JOURNAL;
PROTEIN DETERMINATION;
PROTEIN EXPRESSION;
PROTEIN INDUCTION;
PROTEIN SYNTHESIS INHIBITION;
RAT;
REGULATORY MECHANISM;
SIGNAL TRANSDUCTION;
SUPRAOPTIC NUCLEUS;
ANIMALS;
ANTERIOR HYPOTHALAMIC NUCLEUS;
CALCIUM;
CALCIUM CHANNELS, L-TYPE;
CYCLIC AMP-DEPENDENT PROTEIN KINASES;
HYPOTHALAMO-HYPOPHYSEAL SYSTEM;
MALE;
MORPHINE;
NALOXONE;
PARAVENTRICULAR HYPOTHALAMIC NUCLEUS;
PITUITARY-ADRENAL SYSTEM;
PROTEIN KINASE C;
PROTO-ONCOGENE PROTEINS C-FOS;
RATS;
RATS, SPRAGUE-DAWLEY;
SIGNAL TRANSDUCTION;
SUBSTANCE WITHDRAWAL SYNDROME;
SUPRAOPTIC NUCLEUS;
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EID: 0035955352
PISSN: 00142999
EISSN: None
Source Type: Journal
DOI: 10.1016/S0014-2999(01)01356-5 Document Type: Article |
Times cited : (13)
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References (49)
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