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Proceedings of the National Academy of Sciences of the United States of America

Volumn 98, Issue 26, 2001, Pages 14985-14990

Granzyme B can cause mitochondrial depolarization and cell death in the absence of BID, BAX, and BAK

(5) Thomas, Dori A a Scorrano, Luca c Putcha, Girish V b Korsmeyer, Stanley J c Ley, Timothy J a,d

a USA (United States)

b WASHINGTON UNIVERSITY SCHOOL OF MEDICINE (United States)

c HOWARD HUGHES MEDICAL INSTITUTE (United States)

d NONE (United States)

Author keywords

[No Author keywords available]

Indexed keywords

CASPASE 8; CYTOCHROME C; GRANZYME B; PERFORIN; PROTEIN BAK; PROTEIN BAX; PROTEIN BCL 2; PROTEIN BID;

ANIMAL CELL; APOPTOSIS; ARTICLE; CELL DEATH; CELLULAR DISTRIBUTION; CONTROLLED STUDY; CYTOTOXIC T LYMPHOCYTE; IMMUNOFLUORESCENCE; KNOCKOUT GENE; LIVER MITOCHONDRION; MEMBRANE DEPOLARIZATION; MEMBRANE POTENTIAL; MITOCHONDRIAL MEMBRANE; MIXED LYMPHOCYTE CULTURE; MOUSE; NICK END LABELING; NONHUMAN; PRIORITY JOURNAL; WESTERN BLOTTING;

ANIMALS; APOPTOSIS; BCL-2 HOMOLOGOUS ANTAGONIST-KILLER PROTEIN; BCL-2-ASSOCIATED X PROTEIN; BH3 INTERACTING DOMAIN DEATH AGONIST PROTEIN; CARRIER PROTEINS; CELL MEMBRANE PERMEABILITY; CYTOCHROME C GROUP; FLUORESCENT ANTIBODY TECHNIQUE; GRANZYMES; MEMBRANE PROTEINS; MICE; MICE, INBRED BALB C; MITOCHONDRIA, LIVER; PROTO-ONCOGENE PROTEINS; PROTO-ONCOGENE PROTEINS C-BCL-2; SERINE ENDOPEPTIDASES; SUBCELLULAR FRACTIONS;

ANIMALIA;

EID: 0035910057 PISSN: 00278424 EISSN: None Source Type: Journal
DOI: 10.1073/pnas.261581498 Document Type: Article

Times cited : (106)

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