A reversible component of mitochondrial respiratory dysfunction in apoptosis can be rescued by exogenous cytochrome c

EMBO Journal

Volumn 20, Issue 4, 2001, Pages 661-671

  Mootha, Vamsi K ^a,b   Wei, Michael C ^a   Buttle, Karolyn F ^c   Scorrano, Luca ^a   Panoutsakopoulou, Vily ^a   Mannella, Carmen A ^c   Korsmeyer, Stanley J ^a  

^a HOWARD HUGHES MEDICAL INSTITUTE   (United States)

^b BRIGHAM AND WOMEN S HOSPITAL   (United States)

^c WADSWORTH CENTER   (United States)

Author keywords

Apoptosis; Cytochrome c; Fas; Mitochondria

Indexed keywords

CALCIUM; CASPASE; CYTOCHROME C; FAS ANTIGEN; REDUCED NICOTINAMIDE ADENINE DINUCLEOTIDE;

ANIMAL EXPERIMENT; ANIMAL TISSUE; APOPTOSIS; ARTICLE; ENZYME ACTIVITY; FEMALE; MEMBRANE PERMEABILITY; MEMBRANE POTENTIAL; MITOCHONDRIAL MEMBRANE; MITOCHONDRIAL RESPIRATION; MOUSE; NONHUMAN; OXIDATION REDUCTION REACTION; OXYGEN CONSUMPTION; PRIORITY JOURNAL;

ANIMALS; ANTIGENS, CD95; APOPTOSIS; CALCIUM; CYTOCHROME C GROUP; FEMALE; MICE; MICE, INBRED C57BL; MITOCHONDRIA; SIGNAL TRANSDUCTION;

ANIMALIA;

EID: 0035865134     PISSN: 02614189     EISSN: None     Source Type: Journal    
DOI: 10.1093/emboj/20.4.661     Document Type: Article

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