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Volumn 20, Issue 47, 2001, Pages 6930-6937
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Genetic disruption of host interferon-γ drastically enhances the metastasis of pancreatic adenocarcinoma through impaired expression of inducible nitric oxide synthase
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Author keywords
Interferon ; Knockout; Metastasis; Nitric oxide; Pancreas
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Indexed keywords
GAMMA INTERFERON;
LIPOPOLYSACCHARIDE;
NITRIC OXIDE SYNTHASE;
ANIMAL EXPERIMENT;
ANIMAL MODEL;
ANIMAL TISSUE;
ANTINEOPLASTIC ACTIVITY;
ARTICLE;
ASCITES;
CANCER INHIBITION;
CELL SECRETION;
CONTROLLED STUDY;
CYTOTOXICITY;
FOOT PAD;
GENE DISRUPTION;
GENE EXPRESSION REGULATION;
IMMUNOSTIMULATION;
ISOGRAFT;
METASTASIS POTENTIAL;
MOUSE;
NONHUMAN;
PANCREAS ADENOCARCINOMA;
PRIORITY JOURNAL;
PROTEIN EXPRESSION;
SUBCUTANEOUS TISSUE;
ADENOCARCINOMA;
ANIMALS;
CELL DIVISION;
INTERFERON TYPE II;
MACROPHAGES;
MICE;
MICE, INBRED C57BL;
MICE, KNOCKOUT;
NEOPLASM METASTASIS;
NITRIC OXIDE;
NITRIC OXIDE SYNTHASE;
NITRIC OXIDE SYNTHASE TYPE II;
PANCREATIC NEOPLASMS;
RNA, NEOPLASM;
TRANSCRIPTION, GENETIC;
TUMOR CELLS, CULTURED;
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EID: 0035909513
PISSN: 09509232
EISSN: None
Source Type: Journal
DOI: 10.1038/sj.onc.1204871 Document Type: Article |
Times cited : (23)
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References (31)
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