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Volumn 40, Issue 15, 2001, Pages 4792-4799
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The cellular response to DNA damage induced by the enediynes C-1027 and neocarzinostatin includes hyperphosphorylation and increased nuclear retention of replication protein A (RPA) and trans inhibition of DNA replication
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Author keywords
[No Author keywords available]
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Indexed keywords
NUCLEAR RETENTION;
CELLS;
DNA;
ENZYME INHIBITION;
PROTEINS;
TUMORS;
BIOCHEMISTRY;
ALKENYLDIALKYNE DERIVATIVE;
ANTIBIOTIC C 1027;
ANTINEOPLASTIC AGENT;
APHIDICOLIN;
REPLICATION PROTEIN A;
UNCLASSIFIED DRUG;
ZINOSTATIN;
ARTICLE;
CELLULAR DISTRIBUTION;
CONTROLLED STUDY;
CYTOTOXICITY;
DNA DAMAGE;
DNA DETERMINATION;
DNA REPLICATION;
DNA STRAND BREAKAGE;
DNA TEMPLATE;
HUMAN;
HUMAN CELL;
PRIORITY JOURNAL;
PROTEIN LOCALIZATION;
PROTEIN PHOSPHORYLATION;
AMINOGLYCOSIDES;
ANTI-BACTERIAL AGENTS;
ANTIBIOTICS, ANTINEOPLASTIC;
BLOTTING, WESTERN;
CELL LINE, TRANSFORMED;
CELL NUCLEUS;
CELL-FREE SYSTEM;
DNA;
DNA DAMAGE;
DNA REPLICATION;
DNA, VIRAL;
DNA-BINDING PROTEINS;
ELECTROPHORESIS, POLYACRYLAMIDE GEL;
ENEDIYNES;
HUMANS;
INTRACELLULAR FLUID;
PHOSPHORYLATION;
REPLICATION PROTEIN A;
SIMIAN VIRUS 40;
SOLUBILITY;
TEMPLATES, GENETIC;
TRANS-ACTIVATION (GENETICS);
ZINOSTATIN;
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EID: 0035901544
PISSN: 00062960
EISSN: None
Source Type: Journal
DOI: 10.1021/bi001668t Document Type: Article |
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Times cited : (21)
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References (46)
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