White matter changes and cognitive performance in aging;Cambios en la sustancia blanca y rendimiento cognitivo en el envejecimiento

Revista de Neurologia

Volumn 33, Issue 4, 2001, Pages 347-353

  Bartres Faz D ^a   Clemente, I C ^a   Junque C ^a  

^a UNIVERSITY OF BARCELONA   (Spain)

Author keywords

Ageing; Cognitive; Genes; Leukoaraiosis; Neuropsychological frontal; Speed of processing; White matter

Indexed keywords

AGED; AGING; BRAIN FUNCTION; CENTRAL NERVOUS SYSTEM; COGNITION; FRONTAL LOBE; HUMAN; MAJOR CLINICAL STUDY; NEUROPSYCHOLOGY; PATHOPHYSIOLOGY; REVIEW; WHITE MATTER;

EID: 0035899159     PISSN: 02100010     EISSN: None     Source Type: Journal    
DOI: 10.33588/rn.3304.2001071     Document Type: Review

References (56)

1. Age-related white matter changes and cognitive impairment
2. Enfermedad de binswanger: Un tipo frecuente de demencia vascular
3. Pathogenesis of leukoaraiosis: A review
4. Leuko-araiosis: An ancient term for a new problem
5. Leukoaraiosis. Reefing rarefaction
6. Fast FLAIR of the brain: The range of appearances in normal subjects and its application to quantification of white-matter disease
7. Further observations on the pathology of subcortical lesions identified on magnetic resonance imaging
8. Pathologic correlates of increased signals of the centrum semiovale on magnetic resonance imaging
9. Clinical features of leukoaraiosis
10. Cognition and white matter hyperintensities in older adult depressed patients
11. Clinical correlates of white matter findings on cranial magnetic resonance imaging of 3301 elderly people: The cardiovascular health study
12. Age-related brain changes associated with motor function in healthy older people
13. Cerebral white matter lesions and depressive symptoms in elderly adults
14. A follow-up study of blood pressure and cerebral white matter lesions
15. Prevalence of cerebral white matter lesions in elderly people: A population based magnetic resonance imaging study. The Rotterdam Scan Study
16. Cerebral white matter lesions, vascular risk factors and cognitive function in a population-based study. The Rotterdam study
17. Presence and severity of cerebral white matter lesions and hypertension, its treatment, and its control. The ARIC study. Atherosclerosis risk in communities study
18. Cognitive and neurologic findings in subjects with diffuse white matter lucencies on computed tomographic scan (leuko-araiosis)
19. Leuko-araiosis on magnetic resonance imaging and speed of mental processing
20. Hypertension in the elderly is associated with white matter lesions and cognitive decline
21. Neuropsychological correlates of white-matter lesions in healthy elderly subjects
22. Incidental brain lesions on magnetic resonance imaging and neurobehavioral functions in the apparently healthy elderly
23. Neuropsychologic correlates of MRI white matter hyperintensities
24. White matter changes in healthy elderly persons correlate with attention and speed of mental processing
25. Cognitive correlates of ventricular enlargement and cerebral white matter lesions on magnetic resonance imaging
26. The effect of white matter hyperintensity volume on brain structure, cognitive performance, and cerebral metabolism of glucose in 51 healthy adults
27. Magnetic resonance imaging, cerebral abnormalities and neuropsychological test performance in elderly hypertensive subjects: A case-control study
28. Incidental white-matter foci on MRI in 'healthy' subjects: Evidence of subtle cognitive dysfunction
29. Association of middle blood pressure to late-life cognitive decline and brain morphology
30. Cerebral white matter lesions and cognitive function: The Rotterdam Scan Study
31. 2 signal intensity on brain MR scans of healthy elderly subjects
32. Neuropsychological test findings in subjects with leukoaraiosis
33. Preservation of normal cognitive functioning in elderly subjects with extensive white-matter lesions of long duration
34. Magnetic resonance imaging white matter lesions and cognitive impairment in hypertensive individuals
35. White-matter hyperintensity and neuropsychological functions in dementia and healthy aging
36. Neuropsychological impairment correlates with hypoperfusion and hypometabolism but not with severity of white matter lesions on MRI in patients with cerebral microangiopathy
37. MRI and genetic correlates of cognitive function in elders with memory impairment
38. Clinical significance of white matter lesions in the elderly
39. Neuropsychological importance of subcortical white matter hyperintensity
40. Cognitive correlates of cerebral white matter changes
41. MRI white matter hyperintensities. Three-year follow-up of the Austrian Prevention Stroke Study
42. Impact of apolipoprotein E ε4 and vascular disease on brain morphology in men from the NHLB1 twin study
43. Changes in cerebral function associated with normal aging
44. Evidence for genetic variance in white matter hyperintensity volume in normal elderly male twins
45. Apolipoprotein E polymorphism and silent micro angiopathy-related cerebral damage. Results of the Austrian stroke prevention study
46. Are genetic factors important in the aetiology of leucoaraiosis? Results from a memory clinic population
47. White matter lesions in Alzheimer patients are influenced by apolipoprotein E genotype
48. Alteración cognitiva en el envejecimiento normal: Nosología y estado actual
49. A semiquantitative rating scale for the assessment of signal hyperintensities on magnetic resonance imaging
50. Apo E influences declarative and procedural learning in age-associated memory impairment
51. Angiotensin I converting enzyme polymorphism in humans with age-associated memory impairment: Relationship with cognitive performance
52. Apolipoprotein E ε4 allele, temporal lobe atrophy, and white matter lesions in late-life dementias
53. Relationship between Apo E, MR1 findings and cognitive function in the cardiovascular health study
54. Angiotensin-converting enzyme insertion/deletion polymorphism and cerebrovascular disease
55. Angiotensin-converting enzyme gene I/D polymorphism and carotid plaques in Japanese
56. Evaluation of genetic risk factors for silent brain infarction