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Oncogene

Volumn 20, Issue 39, 2001, Pages 5503-5510

Functional impact of concomitant versus alternative defects in the Chk2-p53 tumour suppressor pathway

(6) Falck, Jacob a Lukas, Claudia a Protopopova, Marina b Lukas, Jiri a Selivanova, Galina b Bartek, Jiri a

a DANISH CANCER SOCIETY RESEARCH CENTER (Denmark)

b KAROLINSKA INSTITUTE (Sweden)

Author keywords

Cell cycle checkpoints; Chk2; DNA damage; p53; Tumour suppressor

Indexed keywords

CHK2 PROTEIN KINASE; MUTANT PROTEIN; PHOSPHOTRANSFERASE; PROTEIN P53; UNCLASSIFIED DRUG;

ARTICLE; CANCER CELL CULTURE; CANCER INHIBITION; CELL CYCLE; COLON CANCER; COMPLEX FORMATION; CONTROLLED STUDY; DNA DAMAGE; ENZYME ACTIVITY; FAMILIAL CANCER; GENE MUTATION; GENOME; HUMAN; HUMAN CELL; IONIZING RADIATION; PRIORITY JOURNAL; PROTEIN BINDING; PROTEIN DOMAIN; PROTEIN PROTEIN INTERACTION; TRANSACTIVATION;

DNA; DNA DAMAGE; GENES, P53; HUMANS; MUTATION; NEOPLASMS; PROTEIN KINASES; PROTEIN-SERINE-THREONINE KINASES; RADIATION, IONIZING; SIGNAL TRANSDUCTION; TRANS-ACTIVATION (GENETICS); TUMOR CELLS, CULTURED; TUMOR SUPPRESSOR PROTEIN P53;

EID: 0035817764 PISSN: 09509232 EISSN: None Source Type: Journal
DOI: 10.1038/sj.onc.1204811 Document Type: Article

Times cited : (98)

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