Bisphosphonate-induced, hemichannel-mediated, anti-apoptosis through the Src/ERK pathway: A gap junction-independent action of connexin43

Cell Communication and Adhesion

Volumn 8, Issue 4-6, 2001, Pages 377-382

  Plotkin, L I ^a   Bellido, T ^a  

^a UNIVERSITY OF ARKANSAS FOR MEDICAL SCIENCES   (United States)

Author keywords

Apoptosis; Bisphosphonates; Bone; Connexin43; ERKs; Src

Indexed keywords

BISPHOSPHONIC ACID DERIVATIVE; CONNEXIN 43; DYE; MITOGEN ACTIVATED PROTEIN KINASE; PROTEIN TYROSINE KINASE;

ANIMAL CELL; APOPTOSIS; ARTICLE; BIOTINYLATION; CARBOXY TERMINAL SEQUENCE; CELL COMMUNICATION; CELL JUNCTION; CELL SURFACE; CELL SURVIVAL; CHANNEL GATING; CONTROLLED STUDY; DRUG RESPONSE; ENZYME ACTIVATION; FIBROBLAST; GENETIC TRANSFECTION; HUMAN; HUMAN CELL; MOUSE; NONHUMAN; OSTEOBLAST; OSTEOCYTE; POROSITY; PRIORITY JOURNAL; PROTEIN DOMAIN; PROTEIN EXPRESSION; PROTEIN FUNCTION; PROTEIN PROTEIN INTERACTION; REGULATORY MECHANISM; SIGNAL TRANSDUCTION;

ANIMALIA;

EID: 0035754503     PISSN: 10615385     EISSN: None     Source Type: Journal    
DOI: 10.3109/15419060109080757     Document Type: Article

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