Neurodegeneration in the thalamus following neonatal hypoxia-ischemia is programmed cell death

Developmental Neuroscience

Volumn 23, Issue 3, 2001, Pages 186-191

  Northington, Frances J ^a,c   Ferriero, Donna M ^b   Martin, Lee J ^a  

^a JOHNS HOPKINS UNIVERSITY SCHOOL OF MEDICINE   (United States)

^b UNIVERSITY OF CALIFORNIA   (United States)

^c JOHNS HOPKINS HOSPITAL   (United States)

Author keywords

Apoptosis; Bax; Caspase; Cytochrome C oxidase; Fas death receptor; Mitochondria

Indexed keywords

CASPASE 3; CASPASE 8; FAS ANTIGEN; PROTEIN BCL 2;

ANIMAL CELL; ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; APOPTOSIS; BRAIN DEVELOPMENT; BRAIN ISCHEMIA; BRAIN NERVE CELL; BRAIN REGION; CONFERENCE PAPER; CONTROLLED STUDY; ENZYME ACTIVATION; FOREBRAIN; NERVE CELL NECROSIS; NERVE DEGENERATION; NEWBORN; NEWBORN HYPOXIA; NONHUMAN; PRIORITY JOURNAL; PROTEIN EXPRESSION; RAT; THALAMUS;

ANIMALS; ANTIGENS, CD95; APOPTOSIS; BCL-2-ASSOCIATED X PROTEIN; CASPASES; CEREBRAL CORTEX; ELECTRON TRANSPORT COMPLEX IV; HYPOXIA-ISCHEMIA, BRAIN; MITOCHONDRIA; NERVE DEGENERATION; NEURAL PATHWAYS; NEURONS; PROTO-ONCOGENE PROTEINS; PROTO-ONCOGENE PROTEINS C-BCL-2; RATS; THALAMUS;

EID: 0035742794     PISSN: 03785866     EISSN: None     Source Type: Journal    
DOI: 10.1159/000046141     Document Type: Conference Paper

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