High insulin requirements and poor metabolic control do not modify the expression, regulation and PKC mediated activation of the p21ras pathway in PBMC from type II diabetic patients

International Journal of Experimental Diabetes Research

Volumn 2, Issue 1, 2001, Pages 47-54

  Rapoport, M J ^a   Levi, O ^a   Weiss, M ^a   Buchs, A ^a   Ramot, Y ^a   Aharoni, D ^a   Mor, A ^a   Elberg, G ^a   Katz, Y ^a   Weissgarten, J ^a  

^a ASSAF HAROFEH MEDICAL CENTER   (Israel)

Author keywords

hSOS; Insulin resistance; p21ras; PBMC; ras GAP; Type II diabetes

Indexed keywords

GUANOSINE TRIPHOSPHATASE ACTIVATING PROTEIN; INSULIN; MITOGEN ACTIVATED PROTEIN KINASE; ORAL ANTIDIABETIC AGENT; PHORBOL 13 ACETATE 12 MYRISTATE; PROTEIN KINASE C; PROTEIN P21; SOS PROTEIN;

ADULT; AGED; ARTICLE; CELL ACTIVATION; CLINICAL ARTICLE; CONTROLLED STUDY; DIABETIC DIET; DIET THERAPY; DISEASE ASSOCIATION; DISEASE SEVERITY; FEMALE; HUMAN; HUMAN CELL; INSULIN RESISTANCE; MALE; METABOLIC REGULATION; MONONUCLEAR CELL; NON INSULIN DEPENDENT DIABETES MELLITUS; PRIORITY JOURNAL; PROTEIN EXPRESSION; PROTEIN FUNCTION;

AGED; CELL DIVISION; CELLS, CULTURED; DIABETES MELLITUS, TYPE 2; DOSE-RESPONSE RELATIONSHIP, DRUG; ENZYME ACTIVATION; HUMANS; HYPOGLYCEMIC AGENTS; INSULIN; INSULIN RESISTANCE; MIDDLE AGED; MITOGEN-ACTIVATED PROTEIN KINASES; NEUTROPHILS; PROTEIN KINASE C; PROTO-ONCOGENE PROTEINS P21(RAS); TETRADECANOYLPHORBOL ACETATE;

EID: 0035693198     PISSN: 15604284     EISSN: None     Source Type: Journal    
DOI: 10.1155/EDR.2001.47     Document Type: Article

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