Hypothesis: Is obesity an inflammatory condition?

Nutrition

Volumn 17, Issue 11-12, 2001, Pages 974-975

  Lee, Myung Duk ^a   Kim, In Chul ^a  

^a The Catholic University of Korea   (South Korea)

Author keywords

[No Author keywords available]

Indexed keywords

ACETYLCHOLINE; C REACTIVE PROTEIN; CYTOKINE; DOPAMINE; INTERLEUKIN 6; LEPTIN; LONG CHAIN FATTY ACID; NEUROPEPTIDE Y; POLYUNSATURATED FATTY ACID; SEROTONIN; TUMOR NECROSIS FACTOR ALPHA;

BEHAVIOR; BLOOD LEVEL; BODY FAT; BODY MASS; BRAIN DEVELOPMENT; BREAST MILK; CALORIC INTAKE; CARDIOVASCULAR RISK; DIABETES MELLITUS; ENERGY EXPENDITURE; ENVIRONMENTAL FACTOR; EXERCISE; GENETIC SUSCEPTIBILITY; GENOTYPE; HEALTH HAZARD; HYPOTHESIS; INCIDENCE; INFLAMMATION; LEUKOCYTE COUNT; OBESITY; PRADER WILLI SYNDROME; PRIORITY JOURNAL; SHORT SURVEY;

EID: 0035666960     PISSN: 08999007     EISSN: None     Source Type: Journal    
DOI: 10.1016/S0899-9007(01)00701-8     Document Type: Editorial

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17. Subcutaneous adipose tissue releases interleukin-6, but not tumor necrosis factor-a, in vivo
18. Omental and subcutaneous adipose tissues of obese subjects release interleukin-6: Depot difference and regulation by glucocorticoid
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