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Volumn 21, Issue 3, 2001, Pages 840-853

The NH2-terminal coiled-coil domain and tyrosine 177 play important roles in induction of a myeloproliferative disease in mice by Bcr-Abl

Author keywords

[No Author keywords available]

Indexed keywords

BCR ABL PROTEIN;

EID: 0035136290     PISSN: 02707306     EISSN: None     Source Type: Journal    
DOI: 10.1128/MCB.21.3.840-853.2001     Document Type: Article
Times cited : (101)

References (50)
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  • 14
    • 0032827603 scopus 로고    scopus 로고
    • Bcr-Abl with an SH3 deletion retains the ability to induce a myeloproliferative disease in mice, yet c-Abl activated by an SH3 deletion induces only lymphoid malignancy
    • (1999) Mol. Cell. Biol. , vol.19 , pp. 6918-6928
    • Gross, A.W.1    Zhang, X.2    Ren, R.3
  • 15
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    • Expression of interferon consensus sequence binding protein (ICSBP) is downregulated in Bcr-Abl-induced murine chronic myelogenous leukemia-like disease, and forced coexpression of ICSBP inhibits Bcr-Abl-induced myeloproliferative disorder
    • (2000) Mol. Cell. Biol. , vol.20 , pp. 1149-1161
    • Hao, S.X.1    Ren, R.2
  • 21
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    • N-terminal mutations activate the leukemogenic potential of the myristoylated form of c-abl
    • (1989) EMBO J. , vol.8 , pp. 449-456
    • Jackson, P.1    Baltimore, D.2
  • 34
    • 0034661522 scopus 로고    scopus 로고
    • The Grb2 binding site is required for the induction of chronic myeloid leukemia-like disease in mice by the Bcr/Abl tyrosine kinase
    • (2000) Blood , vol.96 , pp. 664-670
    • Million, R.P.1    Van Etten, R.A.2
  • 48
    • 0028501914 scopus 로고
    • Non-globular domains in protein sequences: Automated segmentation using complexity measures
    • (1994) Comput. Chem. , vol.18 , pp. 269-285
    • Wootton, J.C.1
  • 50
    • 0032533620 scopus 로고    scopus 로고
    • Bcr-Abl efficiently induces a myeloproliferative disease and production of excess interleukin-3 and granulocyte-macrophage colony-stimulating factor in mice: A novel model for chronic myelogenous leukemia
    • (1998) Blood , vol.92 , pp. 3829-3840
    • Zhang, X.1    Ren, R.2


* 이 정보는 Elsevier사의 SCOPUS DB에서 KISTI가 분석하여 추출한 것입니다.