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Volumn 21, Issue 5, 2001, Pages 1784-1794
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CAC3 (MSI1) suppression of RAS2G19V is independent of chromatin assembly factor I and mediated by NPR1
a a a a a a a a a |
Author keywords
[No Author keywords available]
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Indexed keywords
CHROMATIN ASSEMBLY FACTOR 1;
CHROMATIN PROTEIN;
CYCLIC AMP;
HISTONE H3;
HISTONE H4;
PROTEIN CAC3P;
PROTEIN NPR1P;
PROTEIN RAS2;
RAS PROTEIN;
RETINOBLASTOMA PROTEIN;
UBIQUITIN;
UNCLASSIFIED DRUG;
ARTICLE;
CHROMATIN STRUCTURE;
GENE EXPRESSION REGULATION;
GENE OVEREXPRESSION;
MOLECULAR STABILITY;
NONHUMAN;
PRIORITY JOURNAL;
PROTEIN LOCALIZATION;
PROTEIN PROTEIN INTERACTION;
REGULATORY MECHANISM;
SACCHAROMYCES CEREVISIAE;
SIGNAL TRANSDUCTION;
STRAIN DIFFERENCE;
SUPPRESSOR GENE;
ALLELES;
CELL NUCLEUS;
CHROMOSOMAL PROTEINS, NON-HISTONE;
CYCLIC AMP;
CYTOPLASM;
DNA;
DNA-BINDING PROTEINS;
FUNGAL PROTEINS;
GALACTOSE;
GENOTYPE;
GLUCOSE;
GREEN FLUORESCENT PROTEINS;
HEAT;
LUMINESCENT PROTEINS;
PHENOTYPE;
PLASMIDS;
PRECIPITIN TESTS;
PROTEIN BINDING;
PROTEIN KINASES;
RAS PROTEINS;
RECOMBINANT FUSION PROTEINS;
SACCHAROMYCES CEREVISIAE PROTEINS;
SIGNAL TRANSDUCTION;
SUPPRESSION, GENETIC;
TIME FACTORS;
TWO-HYBRID SYSTEM TECHNIQUES;
SACCHAROMYCES CEREVISIAE;
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EID: 0035135320
PISSN: 02707306
EISSN: None
Source Type: Journal
DOI: 10.1128/MCB.21.5.1784-1794.2001 Document Type: Article |
Times cited : (29)
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References (54)
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