Learning deficits, but normal development and tumor predisposition, in mice lacking exon 23a of Nf1

Nature Genetics

Volumn 27, Issue 4, 2001, Pages 399-405

  Costa, Rui M ^a   Yang, Tao ^b,c   Huynh, Duong P ^d   Pulst, Stefan M ^d   Viskochil, David H ^e   Silva, Alcino J ^a   Brannan, Camilynn I ^b  

^a UNIVERSITY OF CALIFORNIA   (United States)

^b UNIVERSITY OF FLORIDA COLLEGE OF MEDICINE   (United States)

^c BRIGHAM AND WOMEN S HOSPITAL   (United States)

^d UNIVERSITY OF CALIFORNIA   (United States)

^e UNIVERSITY OF UTAH SCHOOL OF MEDICINE   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

ALTERNATIVE RNA SPLICING; ANIMAL EXPERIMENT; ANIMAL MODEL; ARTICLE; AUTOSOMAL DOMINANT DISORDER; CELL VIABILITY; CONTROLLED STUDY; EXON; GENETIC PREDISPOSITION; LEARNING DISORDER; MALIGNANT TRANSFORMATION; MOUSE; NEUROFIBROMATOSIS; NONHUMAN; PRIORITY JOURNAL; PROTEIN DOMAIN; SIGNAL TRANSDUCTION;

ANIMALS; BASE SEQUENCE; DNA PRIMERS; EXONS; GENES, NEUROFIBROMATOSIS 1; GENETIC PREDISPOSITION TO DISEASE; LEARNING DISORDERS; MICE; NEOPLASMS, EXPERIMENTAL; NEUROFIBROMATOSIS 1; REVERSE TRANSCRIPTASE POLYMERASE CHAIN REACTION;

ANIMALIA; RODENTIA;

EID: 0035074432     PISSN: 10614036     EISSN: None     Source Type: Journal    
DOI: 10.1038/86898     Document Type: Article

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