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Volumn 2, Issue 5, 2001, Pages 431-437
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Growth defects induced by perturbation of β1-integrin function in the mammary gland epithelium result from a lack of MAPK activation via the Shc and Akt pathways
a a a a
a
INSTITUT CURIE
(France)
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Author keywords
[No Author keywords available]
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Indexed keywords
BETA1 INTEGRIN;
FOCAL ADHESION KINASE;
GENE PRODUCT;
GROWTH FACTOR RECEPTOR BOUND PROTEIN 2;
MITOGEN ACTIVATED PROTEIN KINASE;
PROTEIN BAD;
PROTEIN KINASE B;
PROTEIN SERINE THREONINE KINASE;
PROTEIN SHC;
STRESS ACTIVATED PROTEIN KINASE;
THREONINE;
TRANSCRIPTION FACTOR;
TRANSCRIPTION FACTOR FKHR;
UNCLASSIFIED DRUG;
ANIMAL CELL;
APOPTOSIS;
ARTICLE;
BREAST EPITHELIUM;
CELL ADHESION;
CELL DIFFERENTIATION;
CELL INTERACTION;
CELL PROLIFERATION;
CELL SURVIVAL;
CONTROLLED STUDY;
EPITHELIUM CELL;
EXTRACELLULAR MATRIX;
FEMALE;
GROWTH REGULATION;
IN VIVO STUDY;
LACTATION;
MITOSIS;
MOUSE;
MUTANT;
NONHUMAN;
PREGNANCY;
PRIORITY JOURNAL;
PROTEIN PHOSPHORYLATION;
SIGNAL TRANSDUCTION;
TRANSGENIC MOUSE;
ANIMALIA;
MUS MUSCULUS;
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EID: 0034986471
PISSN: 1469221X
EISSN: None
Source Type: Journal
DOI: 10.1093/embo-reports/kve086 Document Type: Article |
Times cited : (55)
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References (19)
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