Mitochondrial KATP channel activation reduces anoxic injury by restoring mitochondrial membrane potential

American Journal of Physiology - Heart and Circulatory Physiology

Volumn 281, Issue 3 50-3, 2001, Pages

  Xu, M ^a   Wang, Y ^a   Ayub, A ^a   Ashraf, M ^a  

^a UNIVERSITY OF CINCINNATI COLLEGE OF MEDICINE   (United States)

Author keywords

Apoptosis; ATP; Cytochreme c; Myocytes; Permeability transition pore

Indexed keywords

5 HYDROXYDECANOIC ACID; ADENOSINE TRIPHOSPHATE; CLAMIKALANT; CYCLOSPORIN A; CYTOCHROME C; DIAZOXIDE; POTASSIUM CHANNEL; POTASSIUM CHANNEL BLOCKING AGENT; POTASSIUM ION; UNCLASSIFIED DRUG;

ANIMAL CELL; ANOXIA; APOPTOSIS; ARTICLE; CELL VIABILITY; CONTROLLED STUDY; ENZYME LOCALIZATION; HEART MITOCHONDRION; HEART MUSCLE CELL; HEART MUSCLE FIBER MEMBRANE POTENTIAL; MEMBRANE PERMEABILITY; MEMBRANE POTENTIAL; NEWBORN; NONHUMAN; PRIORITY JOURNAL; RAT;

EID: 0034821212     PISSN: 03636135     EISSN: None     Source Type: Journal    
DOI: 10.1152/ajpheart.2001.281.3.h1295     Document Type: Article

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