Amyloid-β promotes calcium influx and neurodegeneration via stimulation of L voltage-sensitive calcium channels rather than NMDA channels in cultured neurons

Journal of Alzheimer's Disease

Volumn 3, Issue 5, 2001, Pages 479-483

  Ho, Rebecca ^a   Ortiz, Daniela ^a,b  

^a UNIVERSITY OF MASSACHUSETTS LOWELL   (United States)

^b University of Massachusetts Lowell   (United States)

Author keywords

Alzheimer's disease; Amyloid; Calcium; Calcium channels; Glutamate; Neurodegeneration

Indexed keywords

AMYLOID BETA PROTEIN; CALCIUM CHANNEL BLOCKING AGENT; CALCIUM CHANNEL L TYPE; DIZOCILPINE; N METHYL DEXTRO ASPARTIC ACID; NIMOPRIDINE; REACTIVE OXYGEN METABOLITE; TAU PROTEIN; UNCLASSIFIED DRUG;

ANIMAL CELL; APOPTOSIS; ARTICLE; BIOACCUMULATION; CALCIUM TRANSPORT; CHANNEL GATING; CONTROLLED STUDY; CYTOSOL; MICROTUBULE; MOUSE; NERVE CELL CULTURE; NERVE DEGENERATION; NEUROTOXICITY; NONHUMAN; PRIORITY JOURNAL; PROTEIN PHOSPHORYLATION;

EID: 0034767356     PISSN: 13872877     EISSN: None     Source Type: Journal    
DOI: 10.3233/JAD-2001-3507     Document Type: Article

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