The C5a complement activation peptide increases IL-1β and IL-6 release from amyloid-β primed human monocytes: Implications for Alzheimer's disease

Journal of Neuroimmunology

Volumn 109, Issue 2, 2000, Pages 87-94

  O'Barr, Stephen ^a   Cooper, Neil R ^b  

^a WESTERN UNIVERSITY OF HEALTH SCIENCES   (United States)

^b SCRIPPS RESEARCH INSTITUTE   (United States)

Author keywords

Alzheimer's disease; C5a; Complement; Inflammation; Microglia

Indexed keywords

AMYLOID BETA PROTEIN; CHEMOKINE; COMPLEMENT COMPONENT C5A; INTERLEUKIN 1BETA; INTERLEUKIN 6;

ALZHEIMER DISEASE; ARTICLE; ASTROCYTE; COMPLEMENT ACTIVATION; CONTROLLED STUDY; CYTOKINE RELEASE; HUMAN; HUMAN CELL; IMMUNE RESPONSE; IMMUNOSTIMULATION; INFLAMMATION; MICROGLIA; MONOCYTE; NERVE CELL STIMULATION; NEURITE; PRIORITY JOURNAL;

ALZHEIMER DISEASE; AMYLOID BETA-PROTEIN; CELL LINE; COMPLEMENT 5A; COMPLEMENT ACTIVATION; GENE EXPRESSION; HUMAN; INFLAMMATION; INTERLEUKIN-1; INTERLEUKIN-6; MICROGLIA; MONOCYTES; NF-KAPPA B; SUPPORT, NON-U.S. GOV'T; SUPPORT, U.S. GOV'T, P.H.S.; TRANSCRIPTION, GENETIC;

EID: 0034703179     PISSN: 01655728     EISSN: None     Source Type: Journal    
DOI: 10.1016/S0165-5728(00)00291-5     Document Type: Article

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