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Volumn 275, Issue 38, 2000, Pages 29433-29440
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Retrovirally mediated transfer of a G protein-coupled receptor kinase (GRK) dominant-negative mutant enhances endogenous calcitonin receptor signaling in chinese hamster ovary cells. GRK inhibition enhances expression of receptors and receptor mRNA
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Author keywords
[No Author keywords available]
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Indexed keywords
ADENYLATE CYCLASE;
CALCITONIN RECEPTOR;
CYCLIC AMP;
G PROTEIN COUPLED RECEPTOR;
IODINE 125;
MESSENGER RNA;
PHOSPHATIDYLINOSITIDE;
PHOSPHOTRANSFERASE;
SALCATONIN;
ANIMAL CELL;
ARTICLE;
CHO CELL;
CONTROLLED STUDY;
ENZYME INHIBITION;
GENE TRANSFER;
IMMUNOPRECIPITATION;
MUTANT;
NONHUMAN;
POLYACRYLAMIDE GEL ELECTROPHORESIS;
PRIORITY JOURNAL;
PROTEIN EXPRESSION;
RECEPTOR DOWN REGULATION;
RETROVIRUS;
REVERSE TRANSCRIPTION POLYMERASE CHAIN REACTION;
ANIMALS;
CALCITONIN;
CHO CELLS;
CRICETINAE;
GENE EXPRESSION REGULATION, ENZYMOLOGIC;
GENE TRANSFER TECHNIQUES;
GENETIC VECTORS;
GTP-BINDING PROTEINS;
ISOENZYMES;
MUTATION;
PROTEIN-SERINE-THREONINE KINASES;
RETROVIRIDAE;
SIGNAL TRANSDUCTION;
ANIMALIA;
CRICETINAE;
CRICETULUS GRISEUS;
UNIDENTIFIED RETROVIRUS;
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EID: 0034703053
PISSN: 00219258
EISSN: None
Source Type: Journal
DOI: 10.1074/jbc.M003413200 Document Type: Article |
Times cited : (25)
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References (27)
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