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Volumn 19, Issue 52, 2000, Pages 5973-5981
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A switch from p130Cas/Crk to Gab1/Crk signaling correlates with anchorage independent growth and JNK activation in cells transformed by the Met receptor oncoprotein
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Author keywords
Crk; Cytoskeleton; Gab1; JNK; Met tyrosine kinase; Transformation
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Indexed keywords
FOCAL ADHESION KINASE;
ONCOPROTEIN;
PAXILLIN;
PROTEIN P130;
RECEPTOR PROTEIN;
STRESS ACTIVATED PROTEIN KINASE;
TYROSINE KINASE RECEPTOR;
ANCHORAGE INDEPENDENT GROWTH;
ANIMAL CELL;
ARTICLE;
CELL ADHESION;
CELL CYCLE;
CELL SPREADING;
CELL TRANSFORMATION;
CONTROLLED STUDY;
ENZYME ACTIVATION;
GENE CONTROL;
MOUSE;
NONHUMAN;
PRIORITY JOURNAL;
PROTEIN PHOSPHORYLATION;
PROTEIN PROTEIN INTERACTION;
SIGNAL TRANSDUCTION;
WOUND CLOSURE;
3T3 CELLS;
ACTINS;
ANIMALS;
CELL ADHESION;
CELL DIVISION;
CELL SIZE;
CELL TRANSFORMATION, NEOPLASTIC;
CRK-ASSOCIATED SUBSTRATE PROTEIN;
CYTOSKELETAL PROTEINS;
ENZYME ACTIVATION;
FIBRONECTINS;
FOCAL ADHESION KINASE 1;
FOCAL ADHESION PROTEIN-TYROSINE KINASES;
FOCAL ADHESIONS;
JNK MITOGEN-ACTIVATED PROTEIN KINASES;
MAP KINASE KINASE 4;
MICE;
MITOGEN-ACTIVATED PROTEIN KINASE KINASES;
MUTATION;
ONCOGENE PROTEINS;
PAXILLIN;
PHOSPHOPROTEINS;
PHOSPHORYLATION;
PHOSPHOTYROSINE;
PROTEIN BINDING;
PROTEIN-TYROSINE KINASES;
PROTEINS;
PROTO-ONCOGENE PROTEINS;
PROTO-ONCOGENE PROTEINS C-CRK;
PROTO-ONCOGENE PROTEINS C-MET;
RETINOBLASTOMA-LIKE PROTEIN P130;
SIGNAL TRANSDUCTION;
STRESS FIBERS;
WOUND HEALING;
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EID: 0034619763
PISSN: 09509232
EISSN: None
Source Type: Journal
DOI: 10.1038/sj.onc.1203977 Document Type: Article |
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Times cited : (53)
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References (55)
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