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Volumn 192, Issue 3, 2000, Pages 433-438
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Targeted disruption of the leukotriene B4 receptor in mice reveals its role in inflammation and platelet-activating factor-induced anaphylaxis
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Author keywords
Arachidonic acid; Chemotaxis; Knock out; Neutrophil influx; Sex related
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Indexed keywords
COMPLEMENT COMPONENT C5A;
G PROTEIN COUPLED RECEPTOR;
LEUKOTRIENE B4;
THROMBOCYTE ACTIVATING FACTOR;
ANIMAL CELL;
ANIMAL EXPERIMENT;
ANIMAL MODEL;
ARTICLE;
CALCIUM MOBILIZATION;
CHEMOTAXIS;
CONTROLLED STUDY;
DISEASE MODEL;
FEMALE;
GENE DELETION;
IMMEDIATE TYPE HYPERSENSITIVITY;
INFLAMMATION;
MALE;
MOUSE;
NEUTROPHIL;
NONHUMAN;
PRIORITY JOURNAL;
SEX DIFFERENCE;
ANAPHYLAXIS;
ANIMALS;
ARACHIDONIC ACID;
EAR, EXTERNAL;
FEMALE;
GENE TARGETING;
INFLAMMATION MEDIATORS;
MACROPHAGES, PERITONEAL;
MALE;
MICE;
MICE, INBRED C57BL;
MICE, KNOCKOUT;
NEUTROPHILS;
PERITONEUM;
PLATELET ACTIVATING FACTOR;
RECEPTORS, LEUKOTRIENE B4;
ZYMOSAN;
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EID: 0034618033
PISSN: 00221007
EISSN: None
Source Type: Journal
DOI: 10.1084/jem.192.3.433 Document Type: Article |
Times cited : (156)
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References (28)
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