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Volumn 293, Issue 3, 2000, Pages 207-210
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Caspase-3 activation by β-amyloid and prion protein peptides is independent from their neurotoxic effect
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Author keywords
Alzheimer's disease; Apoptosis; Caspase 3; Cortical culture; Prion related encephalopathies
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Indexed keywords
AMYLOID BETA PROTEIN;
CASPASE 3;
CASPASE INHIBITOR;
DEVD CHO;
PEPTIDE;
PRION PROTEIN;
UNCLASSIFIED DRUG;
Z VAD FMK;
ALZHEIMER DISEASE;
ANIMAL CELL;
APOPTOSIS;
ARTICLE;
BRAIN CORTEX;
BRAIN NERVE CELL;
CONTROLLED STUDY;
ENZYME ACTIVATION;
FETUS;
NERVE CELL CULTURE;
NEUROTOXICITY;
NONHUMAN;
PRIORITY JOURNAL;
RAT;
AMINO ACID CHLOROMETHYL KETONES;
AMYLOID BETA-PROTEIN;
ANIMAL;
APOPTOSIS;
CASPASES;
CELL SURVIVAL;
CELLS, CULTURED;
CEREBRAL CORTEX;
CYSTEINE PROTEINASE INHIBITORS;
DOSE-RESPONSE RELATIONSHIP, DRUG;
ENZYME ACTIVATION;
NEURONS;
OLIGOPEPTIDES;
PEPTIDE FRAGMENTS;
PRIONS;
RATS;
SUPPORT, NON-U.S. GOV'T;
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EID: 0034602580
PISSN: 03043940
EISSN: None
Source Type: Journal
DOI: 10.1016/S0304-3940(00)01532-9 Document Type: Article |
Times cited : (31)
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References (22)
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