Alterations in muscarinic receptor-coupled phosphoinositide hydrolysis and AP-1 activation in Alzheimer's disease cybrid cells

Neurobiology of Aging

Volumn 21, Issue 1, 2000, Pages 31-38

  De Sarno, Patrizia ^a   Bijur, Gautam N ^a   Lu, Rong ^a   Davis, Robert E ^b   Jope, Richard S ^a  

^a UNIVERSITY OF ALABAMA AT BIRMINGHAM   (United States)

^b Migenix Inc   (United States)

Author keywords

Alzheimer's disease; AP 1; Cholinergic signaling; Phosphoinositide

Indexed keywords

CARBACHOL; FLUORIDE SODIUM; IONOMYCIN; MUSCARINIC M3 RECEPTOR; MUSCARINIC RECEPTOR; PHOSPHATIDYLINOSITIDE; PHOSPHOLIPASE C; REACTIVE OXYGEN METABOLITE; TRANSCRIPTION FACTOR AP 1;

ALZHEIMER DISEASE; ARTICLE; CHOLINERGIC SYSTEM; CONTROLLED STUDY; DNA BINDING; ELECTROPHORETIC MOBILITY; HUMAN; HUMAN CELL; HYDROLYSIS; IMMUNOBLOTTING; OXIDATIVE STRESS; PRIORITY JOURNAL; SIGNAL TRANSDUCTION;

ALZHEIMER DISEASE; BUTHIONINE SULFOXIMINE; CARBACHOL; CELLS, CULTURED; CHOLINERGIC AGONISTS; DNA; ENZYME INHIBITORS; GLUTATHIONE; GTP-BINDING PROTEINS; HUMANS; HYBRID CELLS; HYDROGEN PEROXIDE; HYDROLYSIS; MITOCHONDRIA; OXIDATIVE STRESS; PHOSPHATIDYLINOSITOLS; PHOSPHOLIPASE C; PROTEIN KINASE C; REACTIVE OXYGEN SPECIES; RECEPTORS, MUSCARINIC; SIGNAL TRANSDUCTION; TRANSCRIPTION FACTOR AP-1;

EID: 0034594489     PISSN: 01974580     EISSN: None     Source Type: Journal    
DOI: 10.1016/S0197-4580(00)00095-6     Document Type: Article

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