Characterization of airway inflammation after repeated exposures to occupational agents

Journal of Allergy and Clinical Immunology

Volumn 106, Issue 6, 2000, Pages 1163-1170

  Lemière, Catherine ^a   Chaboilliez, Simone ^a   Trudeau, Carole ^a   Taha, Rame ^b   Maghni, Karim ^b   Martin, James G ^b   Hamid, Qutayba ^b  

^a HÔPITAL DU SACRÉ COEUR DE MONTRÉAL   (Canada)

^b MCGILL UNIVERSITY   (Canada)

Author keywords

Eosinophils; Eotaxin; IL 5; Induced sputum; Occupational asthma; Provocation challenges

Indexed keywords

EOTAXIN; INTERLEUKIN 5; METHACHOLINE;

ADULT; ARTICLE; CLINICAL ARTICLE; EOSINOPHIL; FEMALE; FORCED EXPIRATORY VOLUME; HUMAN; INFLAMMATORY CELL; INFLAMMATORY DISEASE; LEUKOCYTE DIFFERENTIAL COUNT; MALE; OCCUPATIONAL ASTHMA; OCCUPATIONAL EXPOSURE; PRIORITY JOURNAL; SPUTUM;

EID: 0034538088     PISSN: 00916749     EISSN: None     Source Type: Journal    
DOI: 10.1067/mai.2000.111235     Document Type: Article

References (33)

1. Changes in the cellular profile of induced plutum after allergen-induced asthmatic responses
2. Kinetics of allergen-induced airway eosinophilic cytokine production and airway inflammation
3. Mechanisms and pathology of occupational asthma
4. Sputum eosinophilia after asthmatic responses induced by isocyanates in sensitized subjects
5. Sputum eosinophils and exhaled nitric oxide during late asthmatic reaction in patients with western red cedar asthma
6. Bronchoalveolar neutrophilia during late asthmatic reactions induced by toluene diisocyanate
7. Eosinophilia in transgenic mice expressing interleukin 5
8. Salmeterol treatment reduces the need for inhaled steroids in steroid dependant asthmatics - A randomised, double-blind placebo-controlled parallel group study
9. Recombinant human interleukin 5 is a selective eosinophil chemoattractant
10. Expression of interleukin (IL)-4 and IL-5 proteins in asthma induced by toluene diisocyanate (TDI)
11. 1998 Revision of the Canadian Asthma Consensus Guidelines. Asthma Consensus Conference Editorial Committee
12. Increases in eotaxin-positive cells in induced sputum from atopic asthmatic subjects after inhalational allergen challenge
13. Allergen-induced recruitment of inflammatory cells in lavage 3 and 24 h after challenge in allergic asthmatic lungs
14. Definition and diagnosis of occupational asthma
15. Validation of an exposure system to particles for the diagnosis of occupational asthma
16. Closed-circuit methodology for inhalation challenge tests with isocyanates
17. Bronchial reactivity to inhaled histamine: A method and clinical survey
18. Histamine dose-response curves in asthma: Reproducibility and sensitivity of different indices to assess response
19. Use of induced sputum cell counts to investigate airway inflammation in asthma
20. Indices of airway inflammation in induced sputum: Reproducibility and validity of cell and fluid-phase measurements
21. Induced sputum: Validity of fluid-phase IL-5 measurement
22. IL-4 and IL-5 mRNA expression in induced sputum of asthmatic subjects: Comparison with bronchial wash
23. Localization of atrial natriuretic peptide mRNA and immunoreactivity in the rat heart and human atrial appendage
24. Occupational challenge tests
25. A case for serial examination of sputum inflammatory cells
26. Potential masking effects of salmeterol on airway inflammation in asthma
27. Increases in airway eosinophils and interleukin-5 with minimal bronchoconstriction during repeated low-dose allergen challenge in atopic asthmatics
28. Increase in non-specific bronchial hyperresponsiveness as an early marker of bronchial response to occupational agents during specific inhalation challenges
29. Development of eosinophilic airway inflammation and airway hyperresponsiveness requires interleukin-5 but not immunoglobulin E or B lymphocytes
30. Anti-interleukin 5 but not anti-IgE prevents airway inflammation and airway hyperresponsiveness
31. Autocrine interaction between IL-5 and IL-1beta mediates altered responsiveness of atopic asthmatic sensitized airway smooth muscle
32. Eosinophil chemotactic chemokines (eotaxin, eotaxin-2, RANTES, monocyte chemoattractant protein-3 [MCP-3], and MCP-4), and C-C chemokine receptor 3 expression in bronchial biopsies from atopic and nonatopic (intrinsic) asthmatics
33. Toluene diisocyanate enhances substance P in sensory neurons innervating the nasal mucosa