Expression of virulence genes in Candida albicans

Advances in Experimental Medicine and Biology

Volumn 485, Issue , 2000, Pages 167-176

  Staib, Peter ^a   Kretschmar, Marianne ^b   Nichterlein, Thomas ^b   Kõhler, Gerwald ^a   Morschhäuser, Joachim ^a  

^a UNIVERSITY OF WÜRZBURG   (Germany)

^b UNIVERSITY OF HEIDELBERG   (Germany)

Author keywords

[No Author keywords available]

Indexed keywords

CANDIDA ALBICANS; CONFERENCE PAPER; DNA FLANKING REGION; GENE ACTIVATION; GENE EXPRESSION; GENE INDUCTION; GENETIC ANALYSIS; NONHUMAN; PRIORITY JOURNAL; PROMOTER REGION; VIRULENCE;

CANDIDA ALBICANS; CANDIDIASIS; HUMANS; VIRULENCE;

CANDIDA ALBICANS;

EID: 0034470380     PISSN: 00652598     EISSN: None     Source Type: Journal    
DOI: None     Document Type: Article

References (36)

1. The emerging fungal threat
2. Candida species and virulence
3. Adherence and receptor relationships in Candida albicans
4. Recognition between Candida albicans and host cells
5. Identification of Candida albicans ALS2 and ALS4 and localization of Als proteins to the fungal cell surface
6. Expression of the Candida albicans gene ALSI in Saccharomyces cerevisiae induces adherence to endothelial and epithelial cells
7. Adhesive and mammalian transglutaminease substrate properties of Candida albicans Hwpl
8. Linkage of adhesion, filamentous growth, and virulence in Candida albicans to a single gene, INT1
9. Candida dubliniensis sp. nov.: Phenotypic and molecular characterization of a novel species associated with oral candidosis in HIV-infected individuals
10. Nonfilamentous C. albicans mutants are avirulent
11. Derepressed hyphal growth and reduced virulence in a VH1 family-related protein phosphatase mutant of the human pathogen Candida albicans
12. High-frequency phenotypic switching in Candida albicans
13. Gene regulation during high-frequency switching in Candida albicans
14. Serum-proteins as nitrogen source for yeast-like fungi
15. Candida albicans proteinase as a virulence factor in the pathogenesis of Candida infections
16. Proteinase production and pathogenicity of Candida albicans II. Virulence for mice of C. albicans strains with different proteinase activity
17. Evidence for a correlation between proteinase secretion and vulvovaginal candidosis
18. Evidence for degradation of gastrointestinal mucin by Candida albicans secretory aspartyl proteinase
19. Degradation of human subendothelial extracellular matrix by proteinase-secreting Candida albicans
20. Cleavage of immunoglobulins by pathogenic yeasts of the genus Candida
21. Degradation of humoral host defense by Candida albicans proteinase
22. Scanning electron microscopy of epidermal adherence and cavitation in murine candidiasis: A role for Candida acid proteinase
23. Multiplicity of genes encoding secreted aspartic proteinases in Candida species
24. Differential regulation of SAP8 and SAP9, which encode two new members of the secreted aspartic proteinase family in Candida albicans
25. Disruption of each of the secreted aspartyl proteinase genes SAP1, SAP2, and SAP3 of Candida albicans attenuates virulence
26. A triple deletion of the secreted aspartyl proteinase genes SAP4, SAP5, and SAP6 of Candida albicans causes attenuated virulence
27. Evidence that members of the secretory aspartyl proteinase gene family, in particular SAP2, are virulence factors for Candida vaginitis
28. Expression of seven members of the gene family encoding secretory aspartyl proteinases in Candida albicans
29. Candida albicans secreted aspartyl proteinases: Isoenzyme pattern is determined by cell type, and levels are determined by environmental factors
30. Differential expression of secreted aspartyl proteinases in a model of human oral candidosis and in patient samples from the oral cavity
31. Dissecting the biology of a pathogen during infection
32. Use of genetic recombination as a reporter of gene expression
33. The non-standard genetic code of Candida spp.: An evolving genetic code or a novel mechanism for adaptation?
34. Overexpression of a cloned IMP dehydrogenase gene of Candida albicans confers resistance to the specific inhibitor mycophenolic acid
35. Host-induced, stage-specific virulence gene activation in Candida albicans during infection