Abnormal glutamate transport function in mutant amyloid precursor protein transgenic mice

Experimental Neurology

Volumn 163, Issue 2, 2000, Pages 381-387

  Masliah, Eliezer ^a,b   Alford, Michael ^a   Mallory, Margaret ^a   Rockenstein, Edward ^a   Moechars, Dieder ^c   Van Leuven, Fred ^c  

^a UNIVERSITY OF CALIFORNIA   (United States)

^b UNIVERSITY OF CALIFORNIA   (United States)

^c DEPARTMENT OF PLANT SYSTEMS BIOLOGY   (Belgium)

Author keywords

Amyloid; Confocal; Excitotoxicity; Glutamate; Synapse; Transgenic

Indexed keywords

AMYLOID PRECURSOR PROTEIN; EXCITATORY AMINO ACID RECEPTOR; GLUTAMIC ACID;

ALZHEIMER DISEASE; AMINO ACID TRANSPORT; ANIMAL TISSUE; ARTICLE; CELL DEATH; GENE EXPRESSION REGULATION; MACROGLIA; MOUSE; NERVE DEGENERATION; NEUROPROTECTION; NONHUMAN; NUCLEOTIDE SEQUENCE; PRIORITY JOURNAL; PROTEIN EXPRESSION; PROTEIN PROCESSING; PROTEIN TRANSPORT; SYNAPTIC TRANSMISSION;

EID: 0034042926     PISSN: 00144886     EISSN: None     Source Type: Journal    
DOI: 10.1006/exnr.2000.7386     Document Type: Article

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