Getting signals crossed in C. elegans

Current Opinion in Genetics and Development

Volumn 10, Issue 5, 2000, Pages 523-528

  Hanna Rose, Wendya ^a   Han, Min ^a  

^a UNIVERSITY OF COLORADO SCHOOL OF MEDICINE   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

PROTEIN KINASE;

CAENORHABDITIS ELEGANS; ENZYME ANALYSIS; ENZYME REGULATION; NONHUMAN; PRIORITY JOURNAL; REPRODUCTION; REVIEW; SIGNAL TRANSDUCTION; STIMULUS RESPONSE;

EID: 0033863292     PISSN: 0959437X     EISSN: None     Source Type: Journal    
DOI: 10.1016/S0959-437X(00)00122-2     Document Type: Review

References (59)

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3. pop-1 encodes an HMG box protein required for the specification of a mesoderm precursor in early C. elegans embryos
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47. lin-35 and lin-53, two genes that antagonize a C. elegans Ras pathway, encode proteins similar to Rb and its binding protein RbAp48
48. C. elegans lin-13 functions as a member of the LIN-35 Rb class of genes involved in vulval development and encodes a protein with zinc fingers and an LXCXE motif
49. The RING finger B-box factor TAM-1 and a retinoblastoma-like protein LIN-35 modulate context dependent gene silencing in Caenorhabditis elegans
50. The lin-15 locus encodes two negative regulators of Caenorhabditis elegans vulval development
51. The Caenorhabditis elegans locus lin-15, a negative regulator of a tyrosine kinase signalling pathway, encodes two different proteins
52. The C. elegans gene lin-36 acts cell autonomously in the lin-35 Rb pathway
53. NURD-complex genes antagonise RAs-induced vulval development in Caenorhabditis elegans
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