Cooperation between Ha-ras and fos or transforming growth factor α overcomes a paradoxic tumor-inhibitory effect of p53 loss in transgenic mouse epidermis

Molecular Carcinogenesis

Volumn 29, Issue 2, 2000, Pages 67-75

  Wang, Xiao Jing ^a   Greenhalgh, David A ^b   Donehower, Lawrence A ^a   Roop, Dennis R ^a  

^a BAYLOR COLLEGE OF MEDICINE   (United States)

^b UNIVERSITY OF GLASGOW   (United Kingdom)

Author keywords

Knockout; Oncogene; Skin; Transgenic; Tumor suppressor

Indexed keywords

PROTEIN P53; TRANSFORMING GROWTH FACTOR ALPHA;

ANIMAL CELL; APOPTOSIS; ARTICLE; CELL DIFFERENTIATION; EPIDERMIS CELL; EPIDERMIS HYPERPLASIA; GENE MUTATION; GENE TARGETING; GROWTH INHIBITION; MALIGNANT TRANSFORMATION; MOUSE; NONHUMAN; ONCOGENE C FOS; ONCOGENE H RAS; PRIORITY JOURNAL; SKIN CARCINOGENESIS; TUMOR GROWTH; TUMOR SUPPRESSOR GENE;

ANIMALS; CARCINOGENS; EPIDERMIS; FEMALE; GENE EXPRESSION REGULATION; GENE EXPRESSION REGULATION, NEOPLASTIC; GENES, FOS; GENES, P53; GENES, RAS; KERATINS; MALE; MICE; MICE, INBRED C57BL; MICE, INBRED ICR; MICE, KNOCKOUT; MICE, TRANSGENIC; PAPILLOMA; SKIN NEOPLASMS; TETRADECANOYLPHORBOL ACETATE; TRANSFORMING GROWTH FACTOR ALPHA;

ANIMALIA; MUS MUSCULUS;

EID: 0033758681     PISSN: 08991987     EISSN: None     Source Type: Journal    
DOI: 10.1002/1098-2744(200010)29:2<67::AID-MC3>3.0.CO;2-2     Document Type: Article

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