SCOPUS 정보 검색 플랫폼

Volumn 60, Issue 22, 2000, Pages 6403-6407

Modulation of mitogen-activated protein kinases and phosphorylation of Bcl-2 by vinblastine represent persistent forms of normal fluctuations at G2-M1

(5) Fan, M a Du, L a Stone, A A a Gilbert, K M a Chambers, T C a

a UNIVERSITY OF ARKANSAS FOR MEDICAL SCIENCES (United States)

Author keywords

[No Author keywords available]

Indexed keywords

MITOGEN ACTIVATED PROTEIN KINASE; PROTEIN BCL 2; RAF PROTEIN; STRESS ACTIVATED PROTEIN KINASE; SYNAPTOPHYSIN; VINBLASTINE;

APOPTOSIS; ARTICLE; CELL CYCLE G2 PHASE; CELL SYNCHRONIZATION; ENZYME INACTIVATION; ENZYME INDUCTION; HUMAN; HUMAN CELL; MICROTUBULE; MITOSIS; PRIORITY JOURNAL; PROTEIN PHOSPHORYLATION; SIGNAL TRANSDUCTION;

ACTIVATING TRANSCRIPTION FACTOR 2; ANTINEOPLASTIC AGENTS, PHYTOGENIC; APOPTOSIS; BCL-X PROTEIN; CYCLIC AMP RESPONSE ELEMENT-BINDING PROTEIN; DOSE-RESPONSE RELATIONSHIP, DRUG; ENZYME ACTIVATION; G2 PHASE; HUMANS; JNK MITOGEN-ACTIVATED PROTEIN KINASES; KB CELLS; MAP KINASE KINASE 4; MAP KINASE SIGNALING SYSTEM; MICROTUBULES; MITOGEN-ACTIVATED PROTEIN KINASE KINASES; MITOGEN-ACTIVATED PROTEIN KINASES; MITOSIS; P38 MITOGEN-ACTIVATED PROTEIN KINASES; PHOSPHORYLATION; PROTO-ONCOGENE PROTEINS C-BCL-2; PROTO-ONCOGENE PROTEINS C-JUN; TRANSCRIPTION FACTORS; VINBLASTINE;

EID: 0033708421 PISSN: 00085472 EISSN: None Source Type: Journal
DOI: None Document Type: Article

Times cited : (64)

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