Oppositely imprinted genes p57(Kip2) and Igf2 interact in a mouse model for Beckwith-Wiedemann syndrome

Genes and Development

Volumn 13, Issue 23, 1999, Pages 3115-3124

  Caspary, Tamara ^a   Cleary, Michele A ^a   Perlman, Elizabeth J ^b   Zhang, Pumin ^c   Elledge, Stephen J ^a,d   Tilghman, Shirley M ^a,d  

^a PRINCETON UNIVERSITY   (United States)

^b JOHNS HOPKINS UNIVERSITY SCHOOL OF MEDICINE   (United States)

^c NATIONAL JEWISH MEDICAL AND RESEARCH CENTER   (United States)

^d BAYLOR COLLEGE OF MEDICINE   (United States)

Author keywords

Beckwith Wiedemann syndrome; Genomic imprinting; H19; Igf2; P57(Kip2)

Indexed keywords

CYCLIN DEPENDENT KINASE INHIBITOR; GROWTH FACTOR;

ABDOMINAL WALL DEFECT; ANIMAL MODEL; ANIMAL TISSUE; ARTICLE; BECKWITH WIEDEMANN SYNDROME; CHROMOSOME 11P; CLEFT PALATE; EMBRYO; GENE EXPRESSION REGULATION; GENE LOSS; GENE MUTATION; GENETIC LINKAGE; GENOME IMPRINTING; KIDNEY DYSPLASIA; KNOCKOUT MOUSE; MOUSE; NONHUMAN; PLACENTA DISORDER; PRENATAL GROWTH; PRIORITY JOURNAL; SKELETON MALFORMATION;

ANIMALS; BECKWITH-WIEDEMANN SYNDROME; BONE AND BONES; BONE DEVELOPMENT; CLEFT PALATE; FEMALE; FETAL DEATH; FETAL PROTEINS; FUNGAL PROTEINS; GENES, LETHAL; GENETIC HETEROGENEITY; GENOMIC IMPRINTING; HUMANS; INSULIN-LIKE GROWTH FACTOR II; KIDNEY; MALE; MICE; MICE, INBRED C57BL; MICE, KNOCKOUT; MICE, MUTANT STRAINS; MICROTUBULE-ASSOCIATED PROTEINS; MUSCLE PROTEINS; ORGAN SIZE; PHENOTYPE; PLACENTA; RNA, UNTRANSLATED; SACCHAROMYCES CEREVISIAE PROTEINS;

ANIMALIA; MAMMALIA;

EID: 0033377772     PISSN: 08909369     EISSN: None     Source Type: Journal    
DOI: 10.1101/gad.13.23.3115     Document Type: Article

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