A maternally methylated CpG island in KvLQT1 is associated with an antisense paternal transcript and loss of imprinting in Beckwith-Wiedemann syndrome

Proceedings of the National Academy of Sciences of the United States of America

Volumn 96, Issue 14, 1999, Pages 8064-8069

  Smilinich, N J ^a   Day, C D ^a   Fitzpatrick, G V ^a   Caldwell, G M ^a   Lossie, A C ^a   Cooper, P R ^a   Smallwood, A C ^a   Joyce, J A ^a   Schofield, P N ^a   Reik, W ^a   Nicholls, R D ^a   Weksberg, R ^a   Driscoll, D J ^a   Maher, E R ^a   Shows, T B ^a   Higgins, M J ^a  

^a ROSWELL PARK CANCER INSTITUTE   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

CELL DNA; COMPLEMENTARY RNA; SOMATOMEDIN B;

ANIMAL TISSUE; ARTICLE; BECKWITH WIEDEMANN SYNDROME; CHROMOSOME 11P; CONTROLLED STUDY; CPG ISLAND; DISEASE ASSOCIATION; DNA DETERMINATION; DNA METHYLATION; GENE EXPRESSION; GENE FUNCTION; GENE ISOLATION; GENOME IMPRINTING; HUMAN; HUMAN CELL; HUMAN TISSUE; INTRON; MOUSE; NONHUMAN; NUCLEOTIDE SEQUENCE; PRIORITY JOURNAL;

EID: 0033529207     PISSN: 00278424     EISSN: None     Source Type: Journal    
DOI: 10.1073/pnas.96.14.8064     Document Type: Article

References (52)