Dimerization of mitochondrial bax is associated with increased drug response in bax-transfected A253 cells

Oncology Research

Volumn 11, Issue 2, 1999, Pages 91-99

  Guo, Bin ^a   Yin, Ming Biao ^a   Tóth, Károly ^a   Cao, Shousong ^a   Azrak, Rami G ^a   Rustum, Youcef M ^a  

^a ROSWELL PARK CANCER INSTITUTE   (United States)

Author keywords

Apoptosis; Bax dimerization; Caspase; Cytochrome c; Drug response

Indexed keywords

BAX PROTEIN, HUMAN; CYTOCHROME C; ONCOPROTEIN; PROTEIN BAX; PROTEIN BCL 2; QUINAZOLINE DERIVATIVE; RALTITREXED; THIOPHENE DERIVATIVE; THYMIDYLATE SYNTHASE; TUMOR PROTEIN;

APOPTOSIS; ARTICLE; CELL CULTURE; DIMERIZATION; DOSE RESPONSE; DRUG ANTAGONISM; DRUG EFFECT; ENZYME ACTIVATION; GENETIC TRANSFECTION; GENETICS; HEAD AND NECK TUMOR; HUMAN; METABOLISM; MITOCHONDRION; SQUAMOUS CELL CARCINOMA;

APOPTOSIS; BCL-2-ASSOCIATED X PROTEIN; CARCINOMA, SQUAMOUS CELL; CYTOCHROME C GROUP; DIMERIZATION; DOSE-RESPONSE RELATIONSHIP, DRUG; ENZYME ACTIVATION; HEAD AND NECK NEOPLASMS; HUMANS; MITOCHONDRIA; NEOPLASM PROTEINS; PROTO-ONCOGENE PROTEINS; PROTO-ONCOGENE PROTEINS C-BCL-2; QUINAZOLINES; THIOPHENES; THYMIDYLATE SYNTHASE; TRANSFECTION; TUMOR CELLS, CULTURED;

EID: 0032617046     PISSN: 09650407     EISSN: None     Source Type: Journal    
DOI: None     Document Type: Article

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