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Parental alcohol consumption, cigarette smoking, and risk of Infant leukemia: A Childrens Cancer Group Study
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Shu X-O, Ross JA, Pendergrass TW, Reaman GH, Lampkin B, Robison LL: Parental alcohol consumption, cigarette smoking, and risk of Infant leukemia: a Childrens Cancer Group Study. J Natl Cancer Inst 1996, 88:24-31.
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6
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0030070668
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High frequency of acute promyelocytic leukemia among Latinos with acute myelold leukemia
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Douer D, Preston-Martin S, Chang E, Nichols PW, Watkins KJ, Levine AM: • High frequency of acute promyelocytic leukemia among Latinos with acute myelold leukemia. Blood 1996, 87:308-313. Among patients with AML, acute promyelocytic leukemia was noted in 24.3% to 37.5% of those of Latin American heritage but in only 6.5% to 8.3% of those from other ethnic groups.
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0028837561
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Proposals for the immunological classification of acute leukemias
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European Group for the Immunological Characterization of Leukemias (EGIL), Bene MC, Castoldi G, Knapp W, Ludwig WD, Matutes E, Orfao A, van't Veer MB: Proposals for the immunological classification of acute leukemias. Leukemia 1995, 9:1783-1786.
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Matutes, E.5
Orfao, A.6
Van't Veer, M.B.7
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0029583934
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Toward a clinically useful classification of the acute leukemias
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Pui C-H, Campana D, Crist WM: Toward a clinically useful classification of the acute leukemias. Leukemia 1995, 9:2154-2157.
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0028905531
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Detection of minimal residual disease in acute leukemia: Methodologic advances and clinical significance
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Campana D, Pui C-H: Detection of minimal residual disease in acute • leukemia: methodologic advances and clinical significance. Blood 1995, 85:1416-1434. A comprehensive review of the advances and clinical significance of various methods in the detection of minimal residual leukemia
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Campana, D.1
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0028845148
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Clinical significance of surface antigen expression in children with acute myelold leukemia: Results of study AML-BFM-87
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Creutzig U, Harbott J, Sperling C, Ritter J, Zimmermann M, Löffler H, Riehm H, Schellong G, Ludwig W-D: Clinical significance of surface antigen expression in children with acute myelold leukemia: results of study AML-BFM-87. Blood 1995, 86:3097-3108.
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Creutzig, U.1
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Löffler, H.6
Riehm, H.7
Schellong, G.8
Ludwig, W.-D.9
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13
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0030020460
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The human homologue of rat NG 2, a chondroitin sulfate proteoglycan, is not expressed on the cell surface of normal hematopoietic cells but is expressed by acute myelold leukemia blasts from poor-prognosis patients with abnormalities of chromosome band 11q23
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Smith FO, Rauch C, Williams DE, March CJ, Arthur D, Hilden J, Lampkin BC, Buckley JD, Buckley CV, Woods WG, Dinndorf PA, Sorensen P, Kersey J, Hammond D, Bernstein ID: The human homologue of rat NG 2, a chondroitin sulfate proteoglycan, is not expressed on the cell surface of normal hematopoietic cells but is expressed by acute myelold leukemia blasts from poor-prognosis patients with abnormalities of chromosome band 11q23. Blood 1996, 87:1123-1133.
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Smith, F.O.1
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Arthur, D.5
Hilden, J.6
Lampkin, B.C.7
Buckley, J.D.8
Buckley, C.V.9
Woods, W.G.10
Dinndorf, P.A.11
Sorensen, P.12
Kersey, J.13
Hammond, D.14
Bernstein, I.D.15
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14
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0030043437
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Human homologue of the rat chondroitin sulfate proteoglycan, NG 2, detected by monoclonal antibody 7.1, identifies childhood acute lymphoblastic leukemias with t(4;11)(q21;q23) or t(11;19)(q23;p13) and MLL gene rearrangements
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Behm FG, Smith FO, Raimondi SC, Pui C-H, Bernstein ID: Human homologue of the rat chondroitin sulfate proteoglycan, NG 2, detected by monoclonal antibody 7.1, identifies childhood acute lymphoblastic leukemias with t(4;11)(q21;q23) or t(11;19)(q23;p13) and MLL gene rearrangements. Blood 1996, 87:1134-1139.
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Behm, F.G.1
Smith, F.O.2
Raimondi, S.C.3
Pui, C.-H.4
Bernstein, I.D.5
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15
-
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0030042827
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Leukemia-associated changes identified by quantitative flow cytometry IV. CD34 overexpression in acute myelogenous leukemia M2 with t(8;21)
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Porwit-MacDonald A, Janossy G, Ivory K, Swirsky D, Peters R, Wheatley K, Walker H, Turker A, Goldstone AH, Burnett A: Leukemia-associated changes identified by quantitative flow cytometry IV. CD34 overexpression in acute myelogenous leukemia M2 with t(8;21). Blood 1996, 87:1162-1169.
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Porwit-MacDonald, A.1
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Ivory, K.3
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Peters, R.5
Wheatley, K.6
Walker, H.7
Turker, A.8
Goldstone, A.H.9
Burnett, A.10
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16
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0029120629
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Immunophenotypic changes between diagnosis and relapse in childhood acute lymphoblastic leukemia
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van Wering ER, Beishuizen A, Roeffen ETJM, van der Linden-Schrever BEM, Verhoeven M-AJ, Hählen K, Hooijkaas H, van Dongen JJM: Immunophenotypic changes between diagnosis and relapse in childhood acute lymphoblastic leukemia. Leukemia 1995, 9:1523-1533.
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Van Wering, E.R.1
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Van Der Linden-Schrever, B.E.M.4
Verhoeven, M.-A.J.5
Hählen, K.6
Hooijkaas, H.7
Van Dongen, J.J.M.8
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17
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0030024373
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Phenotypic changes in acute myeloid leukaemia: Implications in the detection of minimal residual disease
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Macedo A, San Miguel JF, Vidnales MB, López-Berges MC, Garcia-Marcos MA, Gonzalez M, Landolfi C, Orfāo A: Phenotypic changes in acute myeloid leukaemia: implications in the detection of minimal residual disease. J Clin Pathol 1996, 49:15-18.
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Macedo, A.1
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López-Berges, M.C.4
Garcia-Marcos, M.A.5
Gonzalez, M.6
Landolfi, C.7
Orfao, A.8
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19
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0028918255
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Clinical and prognostic significance of chromosomal abnormalities in childhood acute myeloid leukemia de novo
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Martinez-Climent JA, Lane NJ, Rubin CM, Morgan E, Hohnstone HS, Mick R, Murphy SB, Vardiman JW, Larson RA, Le Beau MM, Rowley JD: Clinical and prognostic significance of chromosomal abnormalities in childhood acute myeloid leukemia de novo. Leukemia 1995, 9:95-101.
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Martinez-Climent, J.A.1
Lane, N.J.2
Rubin, C.M.3
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Hohnstone, H.S.5
Mick, R.6
Murphy, S.B.7
Vardiman, J.W.8
Larson, R.A.9
Le Beau, M.M.10
Rowley, J.D.11
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20
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0028860940
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The role of chromosome translocations in T-cell acute leukemia
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Hwang L-Y, Baer RJ: The role of chromosome translocations in T-cell acute leukemia. Curr Opin Immunol 1995, 7:659-664.
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Hwang, L.-Y.1
Baer, R.J.2
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21
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0028860943
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A non-random chromosome abnormality found in precursor-B lineage acute lymphoblastic leukaemia: Dic(9;20)(p1?3;q11)
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Slater R, Smit E, Kroes W, Jotterand Bellomo M, Mühlematter D, Harbott J, Behrendt H, Hählen K, Veerman AJP, Hagemeijer A: A non-random chromosome abnormality found in precursor-B lineage acute lymphoblastic leukaemia: dic(9;20)(p1?3;q11). Leukemia 1995, 9:1613-1619.
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Slater, R.1
Smit, E.2
Kroes, W.3
Jotterand Bellomo, M.4
Mühlematter, D.5
Harbott, J.6
Behrendt, H.7
Hählen, K.8
Veerman, A.J.P.9
Hagemeijer, A.10
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22
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0029135133
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Acute lymphoblastic leukemias with deletion of 11q23 or a novel Inversion (11)(p13q23) lack MLL gene rearrangements and have favorable clinical features
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Raimondi SC, Frestedt JL, Pui C-H, Downing JR, Head DR, Kersey JH, Behm FG: Acute lymphoblastic leukemias with deletion of 11q23 or a novel Inversion (11)(p13q23) lack MLL gene rearrangements and have favorable clinical features. Blood 1995, 86:1881-1886.
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Blood
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Raimondi, S.C.1
Frestedt, J.L.2
Pui, C.-H.3
Downing, J.R.4
Head, D.R.5
Kersey, J.H.6
Behm, F.G.7
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23
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0029897897
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Rearrangement of the MLL gene confers a poor prognosis in childhood acute lymphoblastic leukemia, regardless of presenting age
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Behm FG, Raimondi SC, Frestedt JL, Liu Q, Crist WM, Downing JR, Rivera •• GK, Kersey JH, Pui C-H: Rearrangement of the MLL gene confers a poor prognosis in childhood acute lymphoblastic leukemia, regardless of presenting age. Blood 1996, 87:2870-2877. In a stratified analysis adjusted for age and treatment protocol, MLL gene rearrangement was correlated with an inferior treatment outcome among patients with 11q23 rearrangements. The 4-year event-free survival (± SE) was 10% ± 6.5% for patients with the MLL rearrangement and 64% ± 19% for those with other 11q23 abnormalities (not involving the MLL gene), underscoring the importance of molecular analysis in risk assessment of children with ALL.
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Blood
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, pp. 2870-2877
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Behm, F.G.1
Raimondi, S.C.2
Frestedt, J.L.3
Liu, Q.4
Crist, W.M.5
Downing, J.R.6
Rivera, G.K.7
Kersey, J.H.8
Pui, C.-H.9
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24
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0028875252
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Molecular analysis of infant acute lymphoblastic leukemia: MLL gene rearrangement and reverse transcriptase-polymerase chain reaction for t(4;11)(q21;q23)
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Hilden JM, Frestedt JL, Moore RO, Heerema NA, Arthur DC, Reaman GH, Kersey JH: Molecular analysis of infant acute lymphoblastic leukemia: MLL gene rearrangement and reverse transcriptase-polymerase chain reaction for t(4;11)(q21;q23). Blood 1995, 86:3876-3882.
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Blood
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Hilden, J.M.1
Frestedt, J.L.2
Moore, R.O.3
Heerema, N.A.4
Arthur, D.C.5
Reaman, G.H.6
Kersey, J.H.7
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25
-
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0029045087
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The t(12;21) of acute lymphoblastic leukemia results in a tel-AML1 gene fusion
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Romana SP, Mauchauffé M, Le Coníat M, Chumakov I, Le Paslier D, Berger R, Bernard OA: The t(12;21) of acute lymphoblastic leukemia results in a tel-AML1 gene fusion. Blood 1995, 85:3662-3670.
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Blood
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Romana, S.P.1
Mauchauffé, M.2
Le Coníat, M.3
Chumakov, I.4
Le Paslier, D.5
Berger, R.6
Bernard, O.A.7
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26
-
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0029004541
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Fusion of the TEL gene on 12p13 to the AML1 gene on 21q22 in acute lymphoblastic leukemia
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Golub TR, Barker GF, Bohlander SK, Hiebert SW, Ward DC, Bray-Ward P, Morgan E, Raimondi SC, Rowley JD, Gilliland DG: Fusion of the TEL gene on 12p13 to the AML1 gene on 21q22 in acute lymphoblastic leukemia. Proc Natl Acad Sci U S A 1995, 92:4917-4921.
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Golub, T.R.1
Barker, G.F.2
Bohlander, S.K.3
Hiebert, S.W.4
Ward, D.C.5
Bray-Ward, P.6
Morgan, E.7
Raimondi, S.C.8
Rowley, J.D.9
Gilliland, D.G.10
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27
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13344282725
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TEL/AML1 fusion resulting from a cryptic t(12;21) is the most common genetic lesion in pediatric ALL and defines a subgroup of patients with an excellent prognosis
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Shurtleff SA, Buijs A, Behm FG, Rubnitz JE, Raimondi SC, Hancock ML, •• Chan GC-F, Pui C-H, Grosveld G, Downing JR: TEL/AML1 fusion resulting from a cryptic t(12;21) is the most common genetic lesion in pediatric ALL and defines a subgroup of patients with an excellent prognosis. Leukemia 1995, 9:1985-1989. The authors demonstrate a TEL/AML chimeric transcript encoded by a cryptic t(12;21) in 22% of children with newly diagnosed ALL, making it the most frequent genetic lesion in childhood leukemia. This genetic abnormality appears to define a distinct subgroup of ALL cases with a B-lineage phenotype, between 1 and 10 years of age, having a nonhyperdiploid karyotype, and favorable responses to therapy.
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(1995)
Leukemia
, vol.9
, pp. 1985-1989
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-
Shurtleff, S.A.1
Buijs, A.2
Behm, F.G.3
Rubnitz, J.E.4
Raimondi, S.C.5
Hancock, M.L.6
Chan, G.C.-F.7
Pui, C.-H.8
Grosveld, G.9
Downing, J.R.10
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28
-
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0028805405
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High frequency of t(12;21) in childhood B-Ilneage acute lymphoblastic leukemia
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Romana SP, Poirel H, Leconiat M, Flexor M-A, Mauchauffé M, Jonveaux P, •• Macintyre EA, Berger R, Bernard OA: High frequency of t(12;21) in childhood B-Ilneage acute lymphoblastic leukemia. Blood 1995, 86:4263-4269. TEL/AML fusion was found in eight (22%) of 36 children with B-lineage ALL but in none of 24 adult cases.
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(1995)
Blood
, vol.86
, pp. 4263-4269
-
-
Romana, S.P.1
Poirel, H.2
Leconiat, M.3
Flexor, M.-A.4
Mauchauffé, M.5
Jonveaux, P.6
Macintyre, E.A.7
Berger, R.8
Bernard, O.A.9
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29
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9244221153
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The t12;21 translocation involving TEL and deletion of other TEL alleles: Two frequently associated alterations found in childhood acute lymphoblastic leukemia
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Raynaud S, Cave H, Baens M, Bastard C, Cacheux V, Grosgeorge J, Guidal-Giroux C, Guo C, Vilmer E, Marynen P, Grandchamp B: The t12;21 translocation involving TEL and deletion of other TEL alleles: two frequently associated alterations found in childhood acute lymphoblastic leukemia. Blood 1996, 87:3891-2899.
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Blood
, vol.87
, pp. 3891-12899
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-
Raynaud, S.1
Cave, H.2
Baens, M.3
Bastard, C.4
Cacheux, V.5
Grosgeorge, J.6
Guidal-Giroux, C.7
Guo, C.8
Vilmer, E.9
Marynen, P.10
Grandchamp, B.11
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30
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0030056476
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High incidence of TEL/AML1 fusion resulting from a cryptic t(12;21) in childhood B-lineage acute lymphoblastic leukemia in Taiwan
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in press
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Liang D-C, Chou T-B, Chen J-S, Shurtleff SA, Rubnitz JE, Downing JR, Pui C-H, Shih L-Y: High incidence of TEL/AML1 fusion resulting from a cryptic t(12;21) in childhood B-lineage acute lymphoblastic leukemia in Taiwan. Leukemia 1996, in press.
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Leukemia
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Liang, D.-C.1
Chou, T.-B.2
Chen, J.-S.3
Shurtleff, S.A.4
Rubnitz, J.E.5
Downing, J.R.6
Pui, C.-H.7
Shih, L.-Y.8
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31
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0029056756
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Frequent loss of heterozygosity at the TEL gene locus in acute lymphoblastic leukemia of childhood
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Stegmaier K, Pendse S, Barker GF, Bray-Ward P, Ward DC, Montgomery KT, Krauter KS, Reynolds C, Sklar J, Donnelly M, Bohlander SK, Rowley JD, Sallan SE, Gilliland DG, Golub TR: Frequent loss of heterozygosity at the TEL gene locus in acute lymphoblastic leukemia of childhood. Blood 1995, 86:38-44.
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Blood
, vol.86
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-
Stegmaier, K.1
Pendse, S.2
Barker, G.F.3
Bray-Ward, P.4
Ward, D.C.5
Montgomery, K.T.6
Krauter, K.S.7
Reynolds, C.8
Sklar, J.9
Donnelly, M.10
Bohlander, S.K.11
Rowley, J.D.12
Sallan, S.E.13
Gilliland, D.G.14
Golub, T.R.15
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32
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0029073944
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Does activation of the TAL1 gene occur in a majority of patients with T-cell acute lymphoblastic leukemia? A Pediatric Oncology Group Study
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Bash RO, Hall S, Timmons CF, Crist WM, Amylon M, Smith RG, Baer R: • Does activation of the TAL1 gene occur in a majority of patients with T-cell acute lymphoblastic leukemia? A Pediatric Oncology Group Study. Blood 1995, 86:666-676. Ectopic expression of TAL1 was found in two thirds of T-cell ALL cases, including many that did not have obvious alterations of the gene. The finding suggests that the TAL1 gene plays a prominent role in the development of T-cell ALL.
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(1995)
Blood
, vol.86
, pp. 666-676
-
-
Bash, R.O.1
Hall, S.2
Timmons, C.F.3
Crist, W.M.4
Amylon, M.5
Smith, R.G.6
Baer, R.7
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33
-
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0028922510
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Mutations of the p53 and ras genes in childhood t(1;19)-acute lymphoblastic leukemia
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Kawamura M, Kikuchi A, Kobayashi S, Hanada R, Yamamoto K, Horibe K, Shikano T, Ueda K, Hayashi K, Sekiya T, Hayashi Y: Mutations of the p53 and ras genes in childhood t(1;19)-acute lymphoblastic leukemia. Blood 1995, 85:2546-2552.
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Blood
, vol.85
, pp. 2546-2552
-
-
Kawamura, M.1
Kikuchi, A.2
Kobayashi, S.3
Hanada, R.4
Yamamoto, K.5
Horibe, K.6
Shikano, T.7
Ueda, K.8
Hayashi, K.9
Sekiya, T.10
Hayashi, Y.11
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34
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0030020462
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High incidence of potential p53 inactivation in poor outcome childhood acute lymphoblastic leukemia at diagnosis
-
Marks DI, Kurz BW, Link MP, Ng E, Shuster JJ, Lauer SJ, Brodsky I, Haines DS: High incidence of potential p53 inactivation in poor outcome childhood acute lymphoblastic leukemia at diagnosis. Blood 1996, 87:1155-1161.
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Blood
, vol.87
, pp. 1155-1161
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-
Marks, D.I.1
Kurz, B.W.2
Link, M.P.3
Ng, E.4
Shuster, J.J.5
Lauer, S.J.6
Brodsky, I.7
Haines, D.S.8
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35
-
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0028985436
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p16 Gene homozygous deletions in acute lymphoblastic leukemia
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Quesnel B, Preudhomme C, Philippe N, Vanrumbeke M, Dervite I, Lai JL, • Bauters F, Wattel E, Fenaux P: p16 Gene homozygous deletions in acute lymphoblastic leukemia. Blood 1995, 85:657-663. Homozygous deletion of the p16 gene was found in nine of 63 ALL cases (14%) but not in cases with AML, myelodysplastic syndrome, chronic lymphocytic leukemia, or multiple myeloma.
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(1995)
Blood
, vol.85
, pp. 657-663
-
-
Quesnel, B.1
Preudhomme, C.2
Philippe, N.3
Vanrumbeke, M.4
Dervite, I.5
Lai, J.L.6
Bauters, F.7
Wattel, E.8
Fenaux, P.9
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36
-
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0028988044
-
INK4b/MTS2 in pediatric acute lymphoblastic leukemia
-
INK4b/MTS2 in pediatric acute lymphoblastic leukemia. Blood 1995, 85:2321-2330. In this study, a higher frequency of p16 deletion was found in T-cell ALL than in B-lineage ALL. A p16 deletion appeared to correlate with an inferior treatment outcome.
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(1995)
Blood
, vol.85
, pp. 2321-2330
-
-
Okuda, T.1
Shurtleff, S.A.2
Valentine, M.B.3
Raimondi, S.C.4
Head, D.R.5
Behm, F.6
Curcio-Brint, A.M.7
Liu, Q.8
Pui, C.-H.9
Sherr, C.J.10
Beach, D.11
Look, A.T.12
Downing, J.R.13
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37
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0029008903
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Analysis of a family of cyclin-dependent kinase inhibitors: P15/MTS2/INK4B, p16/MTS1/INK4A, and p18 genes in acute lymphoblastic leukemia of childhood
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