Loss of Cntnap2 Causes Axonal Excitability Deficits, Developmental Delay in Cortical Myelination, and Abnormal Stereotyped Motor Behavior

Cerebral Cortex

Volumn 29, Issue 2, 2019, Pages 586-597

  Scott, Ricardo ^a   Sánchez Aguilera, Alberto ^b   Van Elst, Kim ^c   Lim, Lynette ^b   Dehorter, Nathalie ^b   Bae, Sung Eun ^b   Bartolini, Giorgia ^a,b   Peles, Elior ^d   Kas, Martien J H ^c,e   Bruining, Hilgo ^c   Marín, Oscar ^a,b  

^a MIGUEL HERNÁNDEZ UNIVERSITY   (Spain)

^b KING'S COLLEGE LONDON   (United Kingdom)

^c UNIVERSITY MEDICAL CENTER UTRECHT   (Netherlands)

^d WEIZMANN INSTITUTE OF SCIENCE   (Israel)

^e UNIVERSITY OF GRONINGEN   (Netherlands)

Author keywords

axonal action potentials; Caspr2; GABAergic interneurons; Kv1 family potassium channels; myelin

Indexed keywords

CONTACTIN ASSOCIATED PROTEIN LIKE 2; MEMBRANE PROTEIN; SHAKER POTASSIUM CHANNEL; UNCLASSIFIED DRUG; CNTNAP2 PROTEIN, MOUSE; NERVE PROTEIN;

ACTION POTENTIAL; ADULT; ANIMAL CELL; ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; ARTICLE; AXON; BRAIN HISTOLOGY; CLINICAL RESEARCH; CONTROLLED STUDY; DEVELOPMENTAL DELAY; EXCITATORY POSTSYNAPTIC POTENTIAL; FUNCTIONAL CONNECTIVITY; GABAERGIC SYSTEM; GENE MUTATION; GENETIC VARIATION; GRAY MATTER; INTERNEURON; LOCOMOTION; MOUSE; MYELINATION; NEOCORTEX; NERVE CELL EXCITABILITY; NEUROTRANSMISSION; NEUROTRANSMITTER RELEASE; NONHUMAN; PHENOTYPE; POSTNATAL DEVELOPMENT; POSTSYNAPTIC POTENTIAL; PRIORITY JOURNAL; PYRAMIDAL NERVE CELL; SOCIAL INTERACTION; STEREOTYPY; SYNAPTIC TRANSMISSION; WHITE MATTER; ANIMAL; BRAIN CORTEX; C57BL MOUSE; CORPUS CALLOSUM; DEVELOPMENTAL DISORDER; GENETICS; GROWTH, DEVELOPMENT AND AGING; MALE; METABOLISM; MOTOR DYSFUNCTION; MYELINATED NERVE; PATHOLOGY; PHYSIOLOGY; TRANSGENIC MOUSE;

ACTION POTENTIALS; ANIMALS; AXONS; CEREBRAL CORTEX; CORPUS CALLOSUM; DEVELOPMENTAL DISABILITIES; MALE; MEMBRANE PROTEINS; MICE; MICE, INBRED C57BL; MICE, TRANSGENIC; NERVE FIBERS, MYELINATED; NERVE TISSUE PROTEINS; STEREOTYPIC MOVEMENT DISORDER; SYNAPTIC TRANSMISSION;

EID: 85048079479     PISSN: 10473211     EISSN: 14602199     Source Type: Journal    
DOI: 10.1093/cercor/bhx341     Document Type: Article

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