Postconditioning leads to an increase in protein S-nitrosylation

American Journal of Physiology - Heart and Circulatory Physiology

Volumn 306, Issue 6, 2014, Pages

  Tong, Guang ^a,b   Aponte, Angel M ^b   Kohr, Mark J ^b,c   Steenbergen, Charles ^c   Murphy, Elizabeth ^b   Sun, Junhui ^b  

^a GUANGZHOU GENERAL HOSPITAL OF GUANGZHOU MILITARY COMMAND   (China)

^b NATIONAL INSTITUTES OF HEALTH   (United States)

^c DEPARTMENT OF PATHOLOGY   (United States)

Author keywords

Ischemic postconditioning; Nitric oxide; Protein S nitrosylation; Soluble guanylyl cyclase cGMP

Indexed keywords

1H 1,2,4 OXADIAZOLO[4,3 A]QUINOXALIN 1 ONE; ACONITATE HYDRATASE; ALPHA SMOOTH MUSCLE ACTIN; CREATINE KINASE; ELECTRON TRANSFERRING FLAVOPROTEIN; GLYCERALDEHYDE 3 PHOSPHATE DEHYDROGENASE; KT 5823; MALATE DEHYDROGENASE; MALEIMIDE; MYOGLOBIN; MYOSIN LIGHT CHAIN; N(G) NITROARGININE METHYL ESTER; NITRIC OXIDE; OXOGLUTARATE DEHYDROGENASE; PROTEIN; PROTON TRANSPORTING ADENOSINE TRIPHOSPHATE SYNTHASE; RESIN; S NITROSYLATION PROTEIN; UNCLASSIFIED DRUG; CARBAZOLE DERIVATIVE; PROTEIN S;

ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; ARTICLE; CONTROLLED STUDY; HEART MUSCLE ISCHEMIA; HEART MUSCLE REPERFUSION; HEART PERFUSION; ISCHEMIC POSTCONDITIONING; ISOLATED HEART; MALE; MOUSE; NITROSYLATION; NONHUMAN; PRIORITY JOURNAL; PROTEIN ANALYSIS; PROTEIN MODIFICATION; REPERFUSION INJURY; TWO DIMENSIONAL DIFFERENCE GEL ELECTROPHORESIS; ANIMAL; C57BL MOUSE; DRUG EFFECT; METABOLISM; PROTEIN S-NITROSYLATION; SIGNAL TRANSDUCTION; SOLUBLE GUANYLYL CYCLASE/CGMP;

ISCHEMIC POSTCONDITIONING; NITRIC OXIDE; PROTEIN S-NITROSYLATION; SOLUBLE GUANYLYL CYCLASE/CGMP;

ANIMALS; CARBAZOLES; ISCHEMIC POSTCONDITIONING; MALE; MICE; MICE, INBRED C57BL; MODELS, ANIMAL; NG-NITROARGININE METHYL ESTER; NITRIC OXIDE; PROTEIN S; REPERFUSION INJURY; SIGNAL TRANSDUCTION;

EID: 85047687502     PISSN: 03636135     EISSN: 15221539     Source Type: Journal    
DOI: 10.1152/ajpheart.00660.2013     Document Type: Article

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