Protection through postconditioning or a mitochondria-targeted S-nitrosothiol is unaffected by cardiomyocyte-selective ablation of protein kinase G

Basic Research in Cardiology

Volumn 108, Issue 2, 2013, Pages

  Methner, Carmen ^a   Lukowski, Robert ^b   Grube, Karina ^c   Loga, Florian ^d   Smith, Robin A J ^e   Murphy, Michael P ^f   Hofmann, Franz ^d   Krieg, Thomas ^a  

^a UNIVERSITY OF CAMBRIDGE   (United Kingdom)

^b UNIVERSITY OF TÜBINGEN   (Germany)

^c UNIVERSITY OF GREIFSWALD   (Germany)

^d TECHNICAL UNIVERSITY OF MUNICH   (Germany)

^e UNIVERSITY OF OTAGO   (New Zealand)

^f UNIVERSITY OF CAMBRIDGE   (United Kingdom)

Author keywords

Cardioprotection; Myocytes; Postconditioning; Protein kinase G; S nitrosation

Indexed keywords

1H 1,2,4 OXADIAZOLO[4,3 A]QUINOXALIN 1 ONE; ADENOSINE A2 RECEPTOR AGONIST; ADENOSINE A2B RECEPTOR; BAY 606583; CINACIGUAT; CYCLIC GMP DEPENDENT PROTEIN KINASE; GUANYLATE CYCLASE; NITRIC OXIDE; PHOSPHODIESTERASE V; S NITROSOTHIOL; SILDENAFIL; THIOL; UNCLASSIFIED DRUG; BENZOIC ACID DERIVATIVE; PHOSPHODIESTERASE V INHIBITOR; PIPERAZINE DERIVATIVE; PURINE DERIVATIVE; SULFONE;

ANIMAL CELL; ANIMAL EXPERIMENT; ANIMAL MODEL; ARTICLE; CONTINUOUS INFUSION; CONTROLLED STUDY; ENZYME ACTIVATION; ENZYME INHIBITION; HEART INFARCTION SIZE; HEART MUSCLE CELL; HEART MUSCLE ISCHEMIA; HEART MUSCLE REPERFUSION; HEART PROTECTION; ISCHEMIC POSTCONDITIONING; MITOCHONDRIAL MEMBRANE POTENTIAL; MITOCHONDRION; MOUSE; NITROSATION; NONHUMAN; PROTEIN EXPRESSION; RECEPTOR UPREGULATION; REPERFUSION INJURY; ANIMAL; C57BL MOUSE; DRUG EFFECT; HEART; HEART MUSCLE; IMMUNOHISTOCHEMISTRY; METABOLISM; METHODOLOGY; MOUSE MUTANT; PATHOLOGY; PATHOPHYSIOLOGY; WESTERN BLOTTING;

ANIMALS; BENZOATES; BLOTTING, WESTERN; CYCLIC GMP-DEPENDENT PROTEIN KINASES; HEART; IMMUNOHISTOCHEMISTRY; ISCHEMIC POSTCONDITIONING; MICE; MICE, INBRED C57BL; MICE, KNOCKOUT; MITOCHONDRIA; MYOCARDIAL REPERFUSION INJURY; MYOCARDIUM; MYOCYTES, CARDIAC; PHOSPHODIESTERASE 5 INHIBITORS; PIPERAZINES; PURINES; S-NITROSOTHIOLS; SULFONES;

MLCS; MLOWN;

EID: 84873938984     PISSN: 03008428     EISSN: 14351803     Source Type: Journal    
DOI: 10.1007/s00395-013-0337-1     Document Type: Article

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