Local production of tenascin-C acts as a trigger for monocyte/macrophage recruitment that provokes cardiac dysfunction

Cardiovascular Research

Volumn 114, Issue 1, 2018, Pages 123-137

  Abbadi, Dounia ^a   Laroumanie, Fanny ^a   Bizou, Mathilde ^a   Pozzo, Joffrey ^a,b   Daviaud, Danièle ^a   Delage, Christine ^c   Calise, Denis ^c   Gaits Iacovoni, Frederique ^a   Dutaur, Marianne ^a   Tortosa, Florence ^a   Renaud Gabardos, Edith ^a   Douin Echinard, Victorine ^a   Prats, Anne Catherine ^a   Roncalli, Jerome ^a,b   Parini, Angelo ^a   Pizzinat, Nathalie ^a  

^a UNIVERSITÉ DE TOULOUSE   (France)

^b CHU RANGUEIL   (France)

^c Microsurgery Services   (France)

Author keywords

Heart; Macrophages; Monocytes; Tenascin C

Indexed keywords

4 (1 AMINOETHYL) N (4 PYRIDYL)CYCLOHEXANECARBOXAMIDE; ILOMASTAT; LENTIVIRUS VECTOR; RHOA GUANINE NUCLEOTIDE BINDING PROTEIN; TENASCIN; AUTACOID; CHEMOKINE; RHO GUANINE NUCLEOTIDE BINDING PROTEIN; RHO KINASE; RHOA PROTEIN, MOUSE;

ANIMAL EXPERIMENT; ANIMAL MODEL; AORTIC CONSTRICTION; ARTICLE; BONE MARROW TRANSPLANTATION; CARDIAC MUSCLE CELL; CARDIOMYOPATHY; CONTROLLED STUDY; ENZYME ACTIVATION; HEART FIBROBLAST; HEART FUNCTION; HEART TISSUE; HEART VENTRICLE OVERLOAD; HEMATOPOIETIC CELL; INFLAMMATION; MACROPHAGE; MACROPHAGE MIGRATION; MALE; MONOCYTE; MOUSE; NONHUMAN; OUTCOME ASSESSMENT; PHENOTYPE; PRIORITY JOURNAL; PROTEIN EXPRESSION; SIGNAL TRANSDUCTION; ANIMAL; C57BL MOUSE; CARDIAC MUSCLE; CELL MOTION; DEFICIENCY; DISEASE MODEL; FIBROSIS; GENETICS; HEART LEFT VENTRICLE FUNCTION; HEART LEFT VENTRICLE HYPERTROPHY; HEART VENTRICLE REMODELING; KNOCKOUT MOUSE; METABOLISM; PATHOLOGY; PATHOPHYSIOLOGY; TUMOR MICROENVIRONMENT;

ANIMALS; CELL MOVEMENT; CELLULAR MICROENVIRONMENT; CHEMOKINES; DISEASE MODELS, ANIMAL; FIBROSIS; HYPERTROPHY, LEFT VENTRICULAR; INFLAMMATION MEDIATORS; MACROPHAGES; MALE; MICE, INBRED C57BL; MICE, KNOCKOUT; MONOCYTES; MYOCARDIUM; PHENOTYPE; RHO GTP-BINDING PROTEINS; RHO-ASSOCIATED KINASES; SIGNAL TRANSDUCTION; TENASCIN; VENTRICULAR DYSFUNCTION, LEFT; VENTRICULAR FUNCTION, LEFT; VENTRICULAR REMODELING;

EID: 85040639439     PISSN: 00086363     EISSN: 17553245     Source Type: Journal    
DOI: 10.1093/cvr/cvx221     Document Type: Article

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