Pathogenic role of the damage-associated molecular patterns S100A8 and S100A9 in Coxsackievirus B3-induced myocarditis

Circulation: Heart Failure

Volumn 10, Issue 11, 2017, Pages

  Müller, Irene ^a,b   Vogl, Thomas ^c   Pappritz, Kathleen ^a,b   Miteva, Kapka ^a   Savvatis, Konstantinos ^a,d,e   Rohde, David ^f   Most, Patrick ^b,f   Lassner, Dirk ^g   Pieske, Burkert ^a,b,h   Kühl, Uwe ^a   Van Linthout, Sophie ^a,b   Tschöpe, Carsten ^a,b  

^a CHARITÉ UNIVERSITÄTSMEDIZIN BERLIN   (Germany)

^b DZHK GERMAN CENTRE FOR CARDIOVASCULAR RESEARCH   (Germany)

^c UNIVERSITY OF MÜNSTER   (Germany)

^d ST BARTHOLOMEW S HOSPITAL   (United Kingdom)

^e QUEEN MARY UNIVERSITY OF LONDON   (United Kingdom)

^f UNIVERSITY OF HEIDELBERG   (Germany)

^g Institut Kardiale Diagnostik und Therapie (IKDT)   (Germany)

^h DEUTSCHES HERZZENTRUM BERLIN   (Germany)

Author keywords

Coxsackievirus B3; Myocarditis; RAGE; S100A8; S100A9

Indexed keywords

ALARMIN; CALGRANULIN A; CALGRANULIN B; CHEMOKINE; COMPLEMENTARY DNA; MACROPHAGE INFLAMMATORY PROTEIN 2; REACTIVE OXYGEN METABOLITE; SMALL INTERFERING RNA; ADVANCED GLYCATION END PRODUCT RECEPTOR; AGER PROTEIN, MOUSE; CXCL2 CHEMOKINE; CXCL2 PROTEIN, MOUSE; MESSENGER RNA; S100A8 PROTEIN, MOUSE; S100A9 PROTEIN, MOUSE;

ANIMAL CELL; ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; ARTICLE; CLINICAL ARTICLE; CONTROLLED STUDY; COPY NUMBER VARIATION; COXSACKIEVIRUS B3; DOWN REGULATION; ENZYME LINKED IMMUNOSORBENT ASSAY; EXERCISE; GENE AMPLIFICATION; GENE EXPRESSION; GENETIC TRANSFECTION; HEART HEMODYNAMICS; HEART LEFT VENTRICLE FUNCTION; HEART MUSCLE BIOPSY; HISTOLOGY; HUMAN; IMMUNOFLUORESCENCE; INFLAMMATION; KNOCKOUT GENE; KNOCKOUT MOUSE; MACROPHAGE; MONOCYTE; MOUSE; MYOCARDITIS; NEUTROPHIL; NONHUMAN; OXIDATIVE STRESS; PHENOTYPE; PRIORITY JOURNAL; PROTEIN EXPRESSION; RNA ISOLATION; SYSTOLIC BLOOD PRESSURE; WESTERN BLOTTING; ADULT; ANIMAL; C57BL MOUSE; CARDIAC MUSCLE CELL; CASE CONTROL STUDY; COXSACKIE VIRUS INFECTION; DEFICIENCY; DISEASE MODEL; ENTEROVIRUS B; FEMALE; FIBROSIS; GENETICS; HOST PATHOGEN INTERACTION; MALE; METABOLISM; MIDDLE AGED; NEUTROPHIL CHEMOTAXIS; PATHOGENICITY; PATHOLOGY; RAW 264.7 CELL LINE; RNA INTERFERENCE; SIGNAL TRANSDUCTION; VIROLOGY;

ADULT; ANIMALS; CALGRANULIN A; CALGRANULIN B; CASE-CONTROL STUDIES; CHEMOKINE CXCL2; COXSACKIEVIRUS INFECTIONS; DISEASE MODELS, ANIMAL; ENTEROVIRUS B, HUMAN; FEMALE; FIBROSIS; HOST-PATHOGEN INTERACTIONS; HUMANS; MACROPHAGES; MALE; MICE; MICE, INBRED C57BL; MICE, KNOCKOUT; MIDDLE AGED; MYOCARDITIS; MYOCYTES, CARDIAC; NEUTROPHIL INFILTRATION; OXIDATIVE STRESS; RAW 264.7 CELLS; RECEPTOR FOR ADVANCED GLYCATION END PRODUCTS; RNA INTERFERENCE; RNA, MESSENGER; SIGNAL TRANSDUCTION; TRANSFECTION; VENTRICULAR FUNCTION, LEFT;

EID: 85039840034     PISSN: 19413289     EISSN: 19413297     Source Type: Journal    
DOI: 10.1161/CIRCHEARTFAILURE.117.004125     Document Type: Article

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