Sepsis induces telomere shortening: A potential mechanism responsible for delayed pathophysiological events in sepsis survivors?

Molecular Medicine

Volumn 22, Issue , 2016, Pages 886-891

  Oliveira, Naara Mendes ^a   Rios, Ester C S ^a,b   de Lima, Thais Martins ^a   Victorino, Vanessa Jacob ^a   Barbeiro, Hermes ^a   da Silva, Fabiano Pinheiro ^a   Szabo, Csaba ^b   Soriano, Francisco Garcia ^a  

^a UNIVERSITY OF SÃO PAULO   (Brazil)

^b University of Texas Medical Branch School of Medicine   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

SODIUM CHLORIDE; SUPEROXIDE DISMUTASE; THIOBARBITURIC ACID REACTIVE SUBSTANCE;

AGING; ANIMAL EXPERIMENT; ANIMAL TISSUE; ARTICLE; BLOOD SAMPLING; COGNITIVE DEFECT; CONTROLLED STUDY; ENDOTOXEMIA; ENZYME ACTIVITY; GENE EXPRESSION REGULATION; HOSPITAL READMISSION; LIPID PEROXIDATION; MALE; MORTALITY; MOUSE; NONHUMAN; OXIDATIVE STRESS; PATHOPHYSIOLOGY; PHENOTYPE; QUANTITATIVE ANALYSIS; REAL TIME POLYMERASE CHAIN REACTION; SEPSIS; SURVIVOR; TELOMERE SHORTENING; WESTERN BLOTTING;

EID: 85014397202     PISSN: 10761551     EISSN: None     Source Type: Journal    
DOI: 10.2119/molmed.2016.00225     Document Type: Article

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