Correction to: ATP6V1H Deficiency Impairs Bone Development through Activation of MMP9 and MMP13 (PLOS Genetics, (2017), 13, 2, (e1006481), 10.1371/journal.pgen.1006481);ATP6V1H Deficiency Impairs Bone Development through Activation of MMP9 and MMP13

PLoS Genetics

Volumn 13, Issue 2, 2017, Pages

  Zhang, Yihan ^a,b   Huang, Haigen ^a   Zhao, Gexin ^b,c   Yokoyama, Tadafumi ^d   Vega, Hugo ^d   Huang, Yan ^d   Sood, Raman ^d   Bishop, Kevin ^d   Maduro, Valerie ^d   Accardi, John ^d   Toro, Camilo ^d   Boerkoel, Cornelius F ^d   Lyons, Karen ^b,c   Gahl, William A ^d   Duan, Xiaohong ^e   Malicdan, May Christine V ^d   Lin, Shuo ^b  

^a PEKING UNIVERSITY   (China)

^b UNIVERSITY OF CALIFORNIA   (United States)

^c UNIVERSITY OF CALIFORNIA   (United States)

^d NATIONAL HUMAN GENOME RESEARCH INSTITUTE   (United States)

^e FOURTH MILITARY MEDICAL UNIVERSITY   (China)

Author keywords

[No Author keywords available]

Indexed keywords

ADENOSINE TRIPHOSPHATASE; CASPASE 9; COLLAGENASE 3; CRISPR ASSOCIATED PROTEIN; GELATINASE B; PROTEIN ATP6V1H; UNCLASSIFIED DRUG; ATP6V1H PROTEIN, HUMAN; MMP9 PROTEIN, HUMAN; PROTON TRANSPORTING ADENOSINE TRIPHOSPHATE SYNTHASE;

ADULT; ANIMAL CELL; ANIMAL EXPERIMENT; ANIMAL MODEL; ARTICLE; BONE CELL; BONE DEFECT; BONE DENSITY; BONE MASS; BONE STRUCTURE; CASE REPORT; EMBRYO; ENZYME ACTIVATION; FEMALE; GENE INACTIVATION; GENE SEGREGATION; GENETIC CONSERVATION; HETEROZYGOSITY; HOMOZYGOSITY; HUMAN; LOSS OF FUNCTION MUTATION; MALE; MIDDLE AGED; NONHUMAN; OSSIFICATION; OSTEOLYSIS; OSTEOPOROSIS; PROTEIN EXPRESSION; SHORT STATURE; VERTEBRA; YOUNG ADULT; ZEBRA FISH; ANIMAL; BIOSYNTHESIS; BONE DEVELOPMENT; CHONDROCYTE; CRISPR CAS SYSTEM; DEFICIENCY; GENETICS; GROWTH, DEVELOPMENT AND AGING; METABOLISM; MOUSE; MUTATION; PATHOLOGY; SIGNAL TRANSDUCTION;

ADULT; ANIMALS; BONE DENSITY; BONE DEVELOPMENT; CHONDROCYTES; CRISPR-CAS SYSTEMS; HUMANS; MATRIX METALLOPROTEINASE 13; MATRIX METALLOPROTEINASE 9; MICE; MUTATION; OSTEOPOROSIS; SIGNAL TRANSDUCTION; VACUOLAR PROTON-TRANSLOCATING ATPASES; ZEBRAFISH;

EID: 85014093770     PISSN: 15537390     EISSN: 15537404     Source Type: Journal    
DOI: 10.1371/journal.pgen.1006624     Document Type: Erratum

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