메뉴 건너뛰기




Volumn 90, Issue 3, 2016, Pages 466-468

Inhibition of NLRP3 inflammasome as a therapeutic intervention in crystal-induced nephropathy

Author keywords

[No Author keywords available]

Indexed keywords

CALCIUM OXALATE; CP 456773; CREATININE; CRYOPYRIN; GLYCOPROTEIN P 15095; INTERLEUKIN 18; INTERLEUKIN 1BETA; INTERLEUKIN 1BETA CONVERTING ENZYME; PROTEIN INHIBITOR; UNCLASSIFIED DRUG; UREA; INFLAMMASOME;

EID: 84994275552     PISSN: 00852538     EISSN: 15231755     Source Type: Journal    
DOI: 10.1016/j.kint.2016.05.003     Document Type: Note
Times cited : (5)

References (9)
  • 1
    • 77950343791 scopus 로고    scopus 로고
    • Pattern recognition receptors and inflammation
    • 1 Takeuchi, O., Akira, S., Pattern recognition receptors and inflammation. Cell 140 (2010), 805–820.
    • (2010) Cell , vol.140 , pp. 805-820
    • Takeuchi, O.1    Akira, S.2
  • 3
    • 84873827527 scopus 로고    scopus 로고
    • Calcium oxalate crystals induce renal inflammation by NLRP3-mediated IL-1β secretion
    • 3 Mulay, S.R., Kulkarni, O.P., Rupanagudi, K.V., et al. Calcium oxalate crystals induce renal inflammation by NLRP3-mediated IL-1β secretion. J Clin Invest 123 (2013), 236–246.
    • (2013) J Clin Invest , vol.123 , pp. 236-246
    • Mulay, S.R.1    Kulkarni, O.P.2    Rupanagudi, K.V.3
  • 4
    • 84977596862 scopus 로고    scopus 로고
    • An NLRP3-specific inflammasome inhibitor attenuates crystal-induced kidney fibrosis in mice
    • 4 Ludwig-Portugall, I., Bartok, E., Dhana, E., et al. An NLRP3-specific inflammasome inhibitor attenuates crystal-induced kidney fibrosis in mice. Kidney Int 90 (2016), 525–539.
    • (2016) Kidney Int , vol.90 , pp. 525-539
    • Ludwig-Portugall, I.1    Bartok, E.2    Dhana, E.3
  • 5
    • 84873408537 scopus 로고    scopus 로고
    • iGLuc: a luciferase-based inflammasome and protease activity reporter
    • 5 Bartok, E., Bauernfeind, F., Khaminets, M.G., et al. iGLuc: a luciferase-based inflammasome and protease activity reporter. Nat Methods 10 (2013), 147–154.
    • (2013) Nat Methods , vol.10 , pp. 147-154
    • Bartok, E.1    Bauernfeind, F.2    Khaminets, M.G.3
  • 6
    • 0037930829 scopus 로고    scopus 로고
    • Glutathione s-transferase omega 1-1 is a target of cytokine release inhibitory drugs and may be responsible for their effect on interleukin-1beta posttranslational processing
    • 6 Laliberte, R.E., Perregaux, D.G., Hoth, L.R., et al. Glutathione s-transferase omega 1-1 is a target of cytokine release inhibitory drugs and may be responsible for their effect on interleukin-1beta posttranslational processing. J Biol Chem 278 (2003), 16567–16578.
    • (2003) J Biol Chem , vol.278 , pp. 16567-16578
    • Laliberte, R.E.1    Perregaux, D.G.2    Hoth, L.R.3
  • 7
    • 84942902112 scopus 로고    scopus 로고
    • Involvement of endoplasmic reticulum stress in angiotensin II-induced NLRP3 inflammasome activation in human renal proximal tubular cells in vitro
    • 7 Wang, J., Wen, Y., Lv, L.L., et al. Involvement of endoplasmic reticulum stress in angiotensin II-induced NLRP3 inflammasome activation in human renal proximal tubular cells in vitro. Acta Pharmacol Sin 36 (2015), 821–830.
    • (2015) Acta Pharmacol Sin , vol.36 , pp. 821-830
    • Wang, J.1    Wen, Y.2    Lv, L.L.3
  • 8
    • 84879596906 scopus 로고    scopus 로고
    • + efflux is the common trigger of NLRP3 inflammasome activation by bacterial toxins and particulate matter
    • + efflux is the common trigger of NLRP3 inflammasome activation by bacterial toxins and particulate matter. Immunity 38 (2013), 1142–1153.
    • (2013) Immunity , vol.38 , pp. 1142-1153
    • Munoz-Planillo, R.1    Kuffa, P.2    Martinez-Colon, G.3
  • 9
    • 78651393239 scopus 로고    scopus 로고
    • A role for mitochondria in NLRP3 inflammasome activation
    • 9 Zhou, R., Yazdi, A.S., Menu, P., Tschopp, J., A role for mitochondria in NLRP3 inflammasome activation. Nature 469 (2011), 221–225.
    • (2011) Nature , vol.469 , pp. 221-225
    • Zhou, R.1    Yazdi, A.S.2    Menu, P.3    Tschopp, J.4


* 이 정보는 Elsevier사의 SCOPUS DB에서 KISTI가 분석하여 추출한 것입니다.