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Hyperglucagonemia in diabetic ketoacidosis. Its prevalence and significance
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Metabolic manifestations of insulin deficiency do not occur without glucagon action
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Glucagon receptor antibody completely suppresses type 1 diabetes phenotype without insulin by disrupting a novel diabetogenic pathway
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Absence of glucagon and insulin action reveals a role for the GLP-1 receptor in endogenous glucose production
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Insulin knockout mice have extended survival but volatile blood glucose levels on leptin therapy
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Evidence that in uncontrolled diabetes, hyperglucagonemia is required for ketosis but not for increased hepatic glucose production or hyperglycemia
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[16] Meek, T.H., Dorfman, M.D., Matsen, M.E., Fischer, J.D., Cubelo, A., Kumar, M.R., et al. Evidence that in uncontrolled diabetes, hyperglucagonemia is required for ketosis but not for increased hepatic glucose production or hyperglycemia. Diabetes 64 (2015), 2376–2387.
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Acute disruption of glucagon secretion or action does not improve glucose tolerance in an insulin-deficient mouse model of diabetes
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Blockade of glucagon signaling prevents or reverses diabetes onset only if residual beta-cells persist
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